Queenie K.‐G. Tan - Concepedia

Author

Queenie K.‐G. Tan

Also Known As

Arjun Tarakad, Camilo Toro, Cecelia P Tamburro, Cecelia P. Tamburro, Dave Viskochil, David A. Sweetser, Edward C. Smith, Holly K. Tabor, Jennifer A. Sullivan, Laurie D. Smith

Publications

1.7K

Citations

H-Index

Concepts

All Affiliations

Baylor College of Medicine Columbia University Boston Children's Hospital Duke University Harvard University

About

Queenie K.‐G. Tan is an author at Baylor College of Medicine specializing in medicine, genetics, and pathology.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	7	8	507
2	Engineering	1	2	160
3	Natural Sciences	1	1	163
4	Social Sciences	1	1	14
5	Biology	1	1	163

Queenie K.‐G. Tan

Publications

1.1K

Citations

H-Index

Page 1

	Year	Citations
An <i>Arabidopsis</i> F-box protein acts as a transcriptional co-factor to regulate floral development Eunyoung Chae, Queenie K.‐G. Tan, Theresa Hill, Development	2008	211
The Arabidopsis Zinc Finger-Homeodomain Genes Encode Proteins with Unique Biochemical Properties That Are Coordinately Expressed during Floral Development Queenie K.‐G. Tan, Vivian F. Irish PLANT PHYSIOLOGY	2006	177
Regulation of<i>APETALA3</i>floral homeotic gene expression by meristem identity genes Rebecca Lamb, Theresa Hill, Queenie K.‐G. Tan, Development BiologyArabidopsis Apetala3Plant Molecular BiologyDyad SymmetryDevelopmental Biology	2002	163
A comprehensive iterative approach is highly effective in diagnosing individuals who are exome negative Vandana Shashi, Kelly Schoch, Rebecca C. Spillmann, Genetics in Medicine EngineeringMedicineComprehensive Iterative ApproachDiagnosisPathology	2018	80
De Novo Variants in WDR37 Are Associated with Epilepsy, Colobomas, Dysmorphism, Developmental Delay, Intellectual Disability, and Cerebellar Hypoplasia Oguz Kanca, Jonathan C. Andrews, Pei-Tseng Lee, The American Journal of Human Genetics Wdr37 Are AssociatedMendelian DisorderGenetic DisorderGeneticsDe Novo Variants	2019	55
An autosomal dominant neurological disorder caused by de novo variants in FAR1 resulting in uncontrolled synthesis of ether lipids Sacha Ferdinandusse, Kirsty McWalter, Heleen te Brinke, Genetics in Medicine	2020	49
Alternative transcripts in variant interpretation: the potential for missed diagnoses and misdiagnoses Kelly Schoch, Queenie K.‐G. Tan, Nicholas Stong, Genetics in Medicine Clinical LanguageGenetic DisorderMedicineGeneticsDifferential Diagnosis	2020	42
Clinical sites of the Undiagnosed Diseases Network: unique contributions to genomic medicine and science Kelly Schoch, Cecilia Esteves, Anna Bican, Genetics in Medicine Undiagnosed Diseases NetworkTranslational BioinformaticsMedicineGeneticsGenetic Epidemiology	2020	39
Bi-allelic Variants in TONSL Cause SPONASTRIME Dysplasia and a Spectrum of Skeletal Dysplasia Phenotypes Lindsay C. Burrage, John J. Reynolds, Nissan Baratang, The American Journal of Human Genetics Allelic VariantGenetic DisorderGeneticsBi-allelic VariantsPathology	2019	34
<i>SPTSSA</i> variants alter sphingolipid synthesis and cause a complex hereditary spastic paraplegia Siddharth Srivastava, Hagar Mor Shaked, Kenneth Gable, Brain	2023	33

Page 1