Concepedia

Author

Jean-Luc Laı̈

Inserm

Also Known As

Jean-Luc La, Jean-Luc Lai, Jean-Luc Laï, Jean-Luc Laï, Jean-Luc Laı̈, Jean‐Luc Lai, Jean‐Luc Laï

58

Publications

2.4K

Citations

20

H-Index

33

Concepts

All Affiliations

InsermCentre Hospitalier Universitaire de LilleInstitut pour la Recherche sur le Cancer de LilleHôpital Jeanne de FlandreHôpital Albert Calmette
JL

About

Jean-Luc Laı̈ is an author at Inserm specializing in hematology, medicine, and pathology.

Top Concepts

#ConceptH-IndexPublicationsCitations

1

Medicine

8

8

176

2

Cell Biology

4

4

96

3

Immunology

4

4

65

4

Health Sciences

2

2

80

5

Natural Sciences

1

1

16

Jean-Luc Laı̈

×

25

Publications

2.1K

Citations

17

H-Index

YearCitations

Several types of mutations of the Abl gene can be found in chronic myeloid leukemia patients resistant to STI571, and they can pre-exist to the onset of treatment

Catherine Roche‐Lestienne, Valérie Soenen, Nathalie Grardel,

Blood

2002

517

Acute Myeloid Leukemia and Myelodysplastic Syndromes Following Essential Thrombocythemia Treated With Hydroxyurea: High Proportion of Cases With 17p Deletion

Yvon Sterkers, Claude Preudhomme, Jean-Luc Laı̈,

Blood

1998

370

Chromosome 13 abnormalities identified by FISH analysis and serum β2-microglobulin produce a powerful myeloma staging system for patients receiving high-dose therapy

Thierry Façon, Hervé Avet‐Loiseau, Gaëlle Guillerm,

Blood

2001

313

Myeloid cell differentiation arrest by miR-125b-1 in myelodysplasic syndrome and acute myeloid leukemia with the t(2;11)(p21;q23) translocation

Marina Bousquet, Cathy Quelen, Roberto Rosati,

The Journal of Experimental Medicine

2008

248

High WT1 Expression After Induction Therapy Predicts High Risk of Relapse and Death in Pediatric Acute Myeloid Leukemia

Hélène Lapillonne, Aline Renneville, Anne Auvrignon,

Journal of Clinical Oncology

2006

162

RUNX1 DNA-binding mutations and RUNX1-PRDM16 cryptic fusions in BCR-ABL+ leukemias are frequently associated with secondary trisomy 21 and may contribute to clonal evolution and imatinib resistance

Catherine Roche‐Lestienne, Lauréline Deluche, Sélim Corm,

Blood

2008

72

Several Cytogenetic Subclones May Be Identified Within Plasma Cells From Patients With Monoclonal Gammopathy of Undetermined Significance, Both at Diagnosis and During the Indolent Course of This Condition

Marc Zandecki, Jean-Luc Laı̈, Franck Geneviève,

Blood

1997

56

Mechanisms of genesis of variant translocation in chronic myeloid leukemia are not correlated with ABL1 or BCR deletion status or response to imatinib therapy

Steven Richebourg, Virginie Éclache, Christine Pérot,

Cancer Genetics and Cytogenetics

2008

51

Hyperdiploidy Is a Common Finding in Monoclonal Gammopathy of Undetermined Significance and Monosomy 13 Is Restricted to These Hyperdiploid Patients

Maud Brousseau, Xavier Leleu, J Gérard,

Clinical Cancer Research

2007

41

Plasma cell growth fraction using Ki‐67 antigen expression identifies a subgroup of multiple myeloma patients displaying short survival within the ISS stage I

Thomas Gastinne, Xavier Leleu, Alain Duhamel,

European Journal Of Haematology

2007

36

Page 1