M Klemsz - Concepedia

Author

M Klemsz

Indiana University – Purdue University Indianapolis

Also Known As

M J Klemsz, M Klemsz, M. J. Klemsz, M. Klemsz, M.J. Klemsz, Micheal Klemsz

Publications

3.8K

Citations

H-Index

Concepts

All Affiliations

Indiana University – Purdue University Indianapolis Bipar Sanford Burnham Prebys Medical Discovery Institute Walther Cancer Foundation University of California, Los Angeles

About

M Klemsz is an author at Indiana University – Purdue University Indianapolis specializing in medicine, gene expression, and cell biology.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	7	7	2.4K
2	Cell Biology	6	6	1.3K
3	Immunology	4	4	686
4	Natural Sciences	2	2	443
5	Cellular Physiology	1	1	88

M Klemsz

Publications

3.1K

Citations

H-Index

Page 1

	Year	Citations
Targeted disruption of the PU.1 gene results in multiple hematopoietic abnormalities. Scott R. McKercher, Bruce E. Torbett, Kenton L. Anderson, The EMBO Journal Laboratory HematologyMultiple Hematopoietic AbnormalitiesGenetic DisorderMedicineGenetics	1996	1.1K
The Ewing's sarcoma EWS/FLI-1 fusion gene encodes a more potent transcriptional activator and is a more powerful transforming gene than FLI-1. Win May, Stephen L. Lessnick, Benjamin S. Braun, Molecular and Cellular Biology Potent Transcriptional ActivatorTranscriptional RegulationSystems BiologyMedicineAberrant Transcription Factor	1993	498
PU.1 recruits a second nuclear factor to a site important for immunoglobulin kappa 3' enhancer activity. Jagan M.R. Pongubala, Sujatha Nagulapalli, M Klemsz, Molecular and Cellular Biology Molecular RegulationImmunoglobulin Kappa 3ImmunologyDna SequencesTranscriptional Regulation	1992	356
Effect of PU.1 phosphorylation on interaction with NF-EM5 and transcriptional activation J. Pongubala, Charles Van Beveren, Sujatha Nagulapalli, Science	1993	278
Hematopoietic lineage- and stage-restricted expression of the ETS oncogene family member PU.1 Robert Hromas, Attilio Orazi, RS Neiman, Blood	1993	210
The transcription factor PU.1 is involved in macrophage proliferation. Antonio Celada, Francesc E. Borràs, Concepció Soler, The Journal of Experimental Medicine ImmunologyPathologyImmunologic MechanismInflammationTranscriptional Regulation	1996	155
Isolation and characterization of Exodus-2, a novel C-C chemokine with a unique 37-amino acid carboxyl-terminal extension Robert Hromas, Chang H. Kim, M Klemsz, The Journal of Immunology	1997	140
The Ewing’s Sarcoma <i>EWS/FLI-1</i> Fusion Gene Encodes a More Potent Transcriptional Activator and is a More Powerful Transforming Gene than <i>FLI-1</i> William A. May, Stephen L. Lessnick, Benjamin S. Braun, Molecular and Cellular Biology Potent Transcriptional ActivatorTranscriptional RegulationSystems BiologyPowerful Transforming GeneMedicine	1993	129
Induction of c-fos and c-myc expression during B cell activation by IL-4 and immunoglobulin binding ligands. M Klemsz, Louis B. Justement, Ed Palmer, The Journal of Immunology C-myc ExpressionPhorbol Myristate AcetateImmunologyB Cell ActivationImmunologic Mechanism	1989	88
Characterization of the ets oncogene family member, fli-1. M Klemsz, Richard A. Maki, T Papayannopoulou, Journal of Biological Chemistry Molecular CharacterizationSystems BiologyOncogenic AgentNatural SciencesGenetics	1993	87

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