Mohd Parvez Khan - Concepedia

Concepedia

Author

Mohd Parvez Khan

Central Drug Research Institute ORCID

Also Known As

M Khan, M. Khan, M. P. Khan, M.P. Khan, Mohd P Khan, Mohd P. Khan, Mohd Parvez Khan, Mohd. P. Khan, Mohd. Parvez Khan

54

Publications

1.9K

Citations

23

H-Index

67

Concepts

All Affiliations

Central Drug Research Institute Sanjay Gandhi Post Graduate Institute of Medical Sciences Council of Scientific and Industrial Research University of Michigan Indian Institute of Technology Kanpur

MP

About

Mohd Parvez Khan is an author at Central Drug Research Institute specializing in medicine, osteoporosis, and physiology.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	17	18	710
2	Health Sciences	8	8	317
3	Engineering	2	2	57
4	Natural Sciences	1	1	17
5	Allergy	1	1	115

Mohd Parvez Khan

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30

Publications

1.9K

Citations

23

H-Index

	Year	Citations
Effect of brain edema on infarct volume in a focal cerebral ischemia model in rats. Teng‐Nan Lin, Yang He, Gang Wu, Stroke	1993	721
Enhanced Immunoprotective Effects by Anti-IL-17 Antibody Translates to Improved Skeletal Parameters Under Estrogen Deficiency Compared With Anti-RANKL and Anti-TNF-α Antibodies Abdul Malik Tyagi, Mohd Nizam Mansoori, Kamini Srivastava, Journal of Bone and Mineral Research T-regulatory CellImmunologyImmune RegulationGynecologyCd4 T Cell Responses	2014	115
Co‐expression of <i>Arabidopsis</i> transcription factor, <i>At<scp>MYB</scp>12</i>, and soybean isoflavone synthase, <i>Gm<scp>IFS</scp>1</i>, genes in tobacco leads to enhanced biosynthesis of isoflavones and flavonols resulting in osteoprotective activity Ashutosh Pandey, Prashant Misra, Mohd Parvez Khan, Plant Biotechnology Journal	2013	114
Pathophysiological Mechanism of Bone Loss in Type 2 Diabetes Involves Inverse Regulation of Osteoblast Function by PGC-1α and Skeletal Muscle Atrogenes: AdipoR1 as a Potential Target for Reversing Diabetes-Induced Osteopenia Mohd Parvez Khan, Abhishek K. Singh, Amit Joharapurkar, Diabetes Metformin ActionCell DeathDb MiceSkeletal Muscle AtrogenesOrthopaedic Surgery	2015	69
Suppressing Mitochondrial Respiration Is Critical for Hypoxia Tolerance in the Fetal Growth Plate Qing Yao, Mohd Parvez Khan, Christophe Merceron, Developmental Cell BiologyDevelopmental BiologyMitochondrial FunctionRedox RegulatorFetal Growth Plate	2019	64
A naturally occurring naringenin derivative exerts potent bone anabolic effects by mimicking oestrogen action on osteoblasts Gaurav Swarnkar, Kunal Sharan, Jawed A. Siddiqui, British Journal of Pharmacology	2012	59
Hypoxia-inducible factor 2α is a negative regulator of osteoblastogenesis and bone mass accrual Christophe Merceron, Kavitha Ranganathan, Elizabeth Wang, Bone Research	2019	51
Skp2 inhibits osteogenesis by promoting ubiquitin–proteasome degradation of Runx2 Gatha Thacker, Yogesh Kumar, Mohd Parvez Khan, Biochimica et Biophysica Acta (BBA) - Molecular Cell Research SclerostinProtein DegradationSkeletal BiologyUbiquitin–proteasome DegradationMedicine	2016	46
The Effects of Synthetic Conjugated Estrogens, A (Cenestin) on Trabecular Bone Structure and Strength in the Ovariectomized Rat Model Nancy E. Lane, J. L. Kumer, S. Majumdar, Osteoporosis International MechanobiologyTissue EngineeringSynthetic Conjugated EstrogensEngineeringOvariectomized Rat Model	2002	44
Orally Active Osteoanabolic Agent GTDF Binds to Adiponectin Receptors, With a Preference for AdipoR1, Induces Adiponectin-Associated Signaling, and Improves Metabolic Health in a Rodent Model of Diabetes Abhishek K. Singh, Amit Joharapurkar, Mohd Parvez Khan, Diabetes Metabolic DisorderAdiponectin-associated SignalingGastrointestinal Peptide HormoneObesityMetabolic Syndrome	2014	40

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