Bethan R. Hawley - Concepedia

Author

Bethan R. Hawley

Bristol Institute for Transfusion Sciences

Also Known As

B. R. Hawley, Bethan R Hawley, Bethan R. Hawley, Bethan R. Hawley (157404)

Publications

286

Citations

H-Index

Concepts

All Affiliations

Bristol Institute for Transfusion Sciences NHS Blood and Transplant University of Bristol Ospedale Maggiore Fondazione IRCCS Ca' Granda Ospedale Maggiore Policlinico

About

Bethan R. Hawley is an author at Bristol Institute for Transfusion Sciences specializing in cell biology, medicine, and developmental biology.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	4	4	157
2	Cell Biology	4	4	157
3	Natural Sciences	2	2	82
4	Health Sciences	1	1	39
5	Biochemistry	1	1	27

Bethan R. Hawley

Publications

235

Citations

H-Index

Page 1

	Year	Citations
Protein Distribution during Human Erythroblast Enucleation In Vitro Amanda J. Bell, Timothy J. Satchwell, Kate J. Heesom, PLoS ONE Developmental BiologyProtein DistributionMedicineNatural SciencesHematology	2013	55
Non-muscle myosin II drives vesicle loss during human reticulocyte maturation Pedro Luís Moura, Bethan R. Hawley, Tosti J. Mankelow, Haematologica	2018	44
Characteristic phenotypes associated with congenital dyserythropoietic anemia (type II) manifest at different stages of erythropoiesis Timothy J. Satchwell, Stéphanie Pellegrin, Paola Bianchi, Haematologica ImmunohematologyGeneticsBlood CellMolecular GeneticsIron Deficiency	2013	39
The cytoskeletal binding domain of band 3 is required for multiprotein complex formation and retention during erythropoiesis Timothy J. Satchwell, Bethan R. Hawley, Andrew Bell, Haematologica Molecular BiologyCytoskeletonCellular PhysiologyCell InteractionCell Signaling	2014	36
Severe Ankyrin-R deficiency results in impaired surface retention and lysosomal degradation of RhAG in human erythroblasts Timothy J. Satchwell, Andrew Bell, Bethan R. Hawley, Haematologica Ankyrin ExpressionMolecular BiologyCytoskeletonRedox BiologyCellular Physiology	2016	27
<scp>CFTR</scp> potentiators partially restore channel function to <scp>A</scp>561<scp>E</scp>‐CFTR, a cystic fibrosis mutant with a similar mechanism of dysfunction as <scp>F</scp>508del‐<scp>CFTR</scp> Yi‐Ting Wang, Jia Liu, Avgi Loizidou, British Journal of Pharmacology	2014	24
CD47 surface stability is sensitive to actin disruption prior to inclusion within the band 3 macrocomplex Kathryn E. Mordue, Bethan R. Hawley, Timothy J. Satchwell, Scientific Reports	2017	10

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