Michele B. Weiss - Concepedia

Author

Michele B. Weiss

University of Maryland, Baltimore

Also Known As

M B Weiss, M Weiss, M. B. Weiss, M. Weiss, Michele B Weiss, Michele B. Weiss, Michele Weiss, Michèle Weiss

Publications

899

Citations

H-Index

Concepts

All Affiliations

University of Maryland, Baltimore Johns Hopkins University Thomas Jefferson University U-M Rogel Cancer Center Sidney Kimmel Cancer Center

About

Michele B. Weiss is an author at University of Maryland, Baltimore specializing in medicine, cell biology, and tumor biology.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	5	5	411
2	Cell Biology	4	4	379
3	Engineering	2	2	193
4	Health Sciences	1	1	32
5	Applied Physiology	1	1	32

Michele B. Weiss

Publications

863

Citations

H-Index

Page 1

	Year	Citations
Knockin of mutant PIK3CA activates multiple oncogenic pathways John P. Gustin, Bedri Karakas, Michele B. Weiss, Proceedings of the National Academy of Sciences Human BreastCancer ResearchSignal TransductionMutant Pik3caSignaling Pathway	2009	173
Global ultraviolet imager (GUVI): measuring composition and energy inputs for the NASA Thermosphere Ionosphere Mesosphere Energetics and Dynamics (TIMED) mission L. J. Paxton, A. B. Christensen, D. C. Humm, Proceedings of SPIE, the International Society for Optical Engineering/Proceedings of SPIE EngineeringSpace OpticSolar-terrestrial InteractionAtmospheric ScienceAstronomical Image Analysis	1999	168
TWIST1 Is an ERK1/2 Effector That Promotes Invasion and Regulates MMP-1 Expression in Human Melanoma Cells Michele B. Weiss, Ethan V. Abel, Melanie M. Mayberry, Cancer Research	2012	151
Deletion of PTEN Promotes Tumorigenic Signaling, Resistance to Anoikis, and Altered Response to Chemotherapeutic Agents in Human Mammary Epithelial Cells Michele Vítolo, Michele B. Weiss, Marta Szmacinski, Cancer Research Chemotherapeutic AgentsSystems BiologyPten FunctionSignaling PathwayBreast Oncology	2009	94
Deletion of p53 in human mammary epithelial cells causes chromosomal instability and altered therapeutic response Michele B. Weiss, Michele Vítolo, Morassa Mohseni, Oncogene Genome InstabilityChromosomal InstabilityMedicineMammary GlandPathology	2010	57
The TWEAK Receptor Fn14 Is a Therapeutic Target in Melanoma: Immunotoxins Targeting Fn14 Receptor for Malignant Melanoma Treatment Hong Zhou, Sühendan Ekmekçioglu, John W. Marks, Journal of Investigative Dermatology Tweak Receptor Fn14MelanomaImmunologyMalignant Melanoma TreatmentImmune Checkpoint Inhibitor	2012	55
CADM1 is a TWIST1-regulated suppressor of invasion and survival Edward J. Hartsough, Michele B. Weiss, Shea A. Heilman, Cell Death and Disease	2019	45
Solution Structure of Zinc- and Calcium-Bound Rat S100B as Determined by Nuclear Magnetic Resonance Spectroscopy<sup>,</sup> Paul T. Wilder, Kristen M. Varney, Michele B. Weiss, Biochemistry	2005	36
Exercise-Induced Monocytosis and Modulation of Monocyte Function Wilfried Bieger, Michele B. Weiss, Gerd Michel, International Journal of Sports Medicine Physical ActivityExercise-induced MonocytosisExercise MedicineInsulin SignalingGlucose Uptake	1980	32
FOXD3 Modulates Migration through Direct Transcriptional Repression of TWIST1 in Melanoma Michele B. Weiss, Ethan V. Abel, Neda Dadpey, Molecular Cancer Research	2014	27

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