G. Tim Bowden - Concepedia

Author

G. Tim Bowden

Also Known As

G T Bowden, G Tim Bowden, G. Bowden, G. T. Bowden, G. Tim Bowden, G.T Bowden, G.T. BOWDEN, G.T. Bowden, G.TIM BOWDEN, G.Tim Bowden

111

Publications

4.7K

Citations

H-Index

Concepts

All Affiliations

University of Arizona Hormel (United States)University of Minnesota Banner - University Medical Center Tucson German Cancer Research Center

About

G. Tim Bowden is an author at University of Arizona specializing in medicine, dermatology, and cell biology.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	27	41	2.6K
2	Natural Sciences	12	12	673
3	Engineering	3	3	105
4	Health Sciences	3	3	181
5	Public Health	1	1	15

G. Tim Bowden

Publications

4.1K

Citations

H-Index

Page 1

	Year	Citations
Activation of the mouse cellular Harvey-ras gene in chemically induced benign skin papillomas Allan Balmain, Martin Ramsden, G. Tim Bowden, Nature InflammationCutaneous BiologyPathologyWound HealingDermatology	1984	533
The mRNA coding for the secreted protease transin is expressed more abundantly in malignant than in benign tumors. L M Matrisian, G. Tim Bowden, Peter Krieg, Proceedings of the National Academy of Sciences Transin RnaBenign TumorsPathologyDermatologyCancer Biology	1986	260
Increased ROS levels contribute to elevated transcription factor and MAP kinase activities in malignantly progressed mouse keratinocyte cell lines Ashok Gupta, Sabine Rosenberger, G. Tim Bowden Carcinogenesis	1999	202
Raf and MEK Protein Kinases Are Direct Molecular Targets for the Chemopreventive Effect of Quercetin, a Major Flavonol in Red Wine Ki Won Lee, Nam Joo Kang, Yong‐Seok Heo, Cancer Research	2008	192
Role of p38 MAP kinases and ERK in mediating ultraviolet-B induced cyclooxygenase-2 gene expression in human keratinocytes Weixing Chen, Qingbo Tang, Melissa Gonzales, Oncogene Signal TransductionCell RegulationReceptor Tyrosine KinaseP38 Map KinasesPhotocarcinogenesis	2001	185
Myricetin Suppresses UVB-Induced Skin Cancer by Targeting Fyn Sung Keun Jung, Ki Won Lee, Sanguine Byun, Cancer Research	2008	163
Epidermal growth factor. Ability of tumor promoter to alter its degradation, receptor affinity and receptor number. Bruce E. Magun, Lynn M. Matrisian, G. Tim Bowden Journal of Biological Chemistry Tumor BiologyTranscriptional RegulationEpidermal Growth FactorTumor PromoterReceptor Affinity	1980	149
Inhibition of Ultraviolet B-induced Activator Protein-1 (AP-1) Activity by Aspirin in AP-1-Luciferase Transgenic Mice Chuanshu Huang, Weiya Ma, David Hanenberger, Journal of Biological Chemistry	1997	141
Detection of mutant Ha-ras genes in chemically initiated mouse skin epidermis before the development of benign tumors. Mark A. Nelson, B W Futscher, T. Kinsella, Proceedings of the National Academy of Sciences Benign TumorsPathologyActivated Ha-ras OncogeneDermatologyTumor Biology	1992	115
Ultraviolet B-induced Activated Protein-1 Activation Does Not Require Epidermal Growth Factor Receptor but Is Blocked by a Dominant Negative PKCλ/ι Chuanshu Huang, Weiya Ma, G. Tim Bowden, Journal of Biological Chemistry Conventional PkcNovel PkcSignal TransductionDominant Negative Pkcλ/ιSignaling Pathway	1996	114

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