Ü. Deniz Dinçer - Concepedia

Author

Ü. Deniz Dinçer

Also Known As

U Deniz Dincer, U Deniz Dinçer, U. D. Dincer, U. Deniz Dincer, U. Deniz Dinçer, U.DENIZ DINCER, U.Deniz Dincer, UD Dincer, Ü. Deniz Dincer, Ü. Deniz Dinçer

Publications

1.8K

Citations

H-Index

Concepts

All Affiliations

Ankara University Indiana University – Purdue University Indianapolis Indiana University School of Medicine Louisiana State University Health Sciences Center New Orleans University of Nebraska Medical Center

ÜD

About

Ü. Deniz Dinçer is an author at Ankara University specializing in physiology, medicine, and health sciences.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	10	10	692
2	Health Sciences	8	8	446
3	Biochemistry	3	3	440
4	Natural Sciences	1	1	224
5	Applied Physiology	1	1	17

Ü. Deniz Dinçer

Publications

1.7K

Citations

H-Index

Page 1

	Year	Citations
Diabetes Increases Formation of Advanced Glycation End Products on Sarco(endo)plasmic Reticulum Ca2+-ATPase Keshore R. Bidasee, Yinong Zhang, Chun Hong Shao, Diabetes Cardiac MuscleGlycobiologyCardiac RelaxationPlasmic Reticulum Ca2+-atpaseCellular Physiology	2004	224
Leptin receptors are expressed in coronary arteries, and hyperleptinemia causes significant coronary endothelial dysfunction Jarrod D. Knudson, Ü. Deniz Dinçer, Cuihua Zhang, American Journal of Physiology-Heart and Circulatory Physiology	2005	191
The Effect of Diabetes on Expression of β1-,β2-, and β3-Adrenoreceptors in Rat Hearts Ü. Deniz Dinçer, Keshore R. Bidasee, Şahika Güner, Diabetes	2001	172
Chronic Diabetes Increases Advanced Glycation End Products on Cardiac Ryanodine Receptors/Calcium-Release Channels Keshore R. Bidasee, Karuna Nallani, Yongqi Yu, Diabetes	2003	138
Impaired capsaicin-induced relaxation of coronary arteries in a porcine model of the metabolic syndrome Ian N. Bratz, Gregory M. Dick, Johnathan D. Tune, American Journal of Physiology-Heart and Circulatory Physiology Coronary ArteriesMetabolic SyndromeCapsaicin-induced RelaxationCardiovascular Translational ResearchCardiovascular Disease	2008	135
Impaired function of coronary BK<sub>Ca</sub>channels in metabolic syndrome Léna Borbouse, Gregory M. Dick, Shinichi Asano, American Journal of Physiology-Heart and Circulatory Physiology	2009	90
Periadventitial adipose tissue impairs coronary endothelial function via PKC-β-dependent phosphorylation of nitric oxide synthase Gregory A. Payne, H. Glenn Bohlen, Ü. Deniz Dinçer, American Journal of Physiology-Heart and Circulatory Physiology Nitric OxideCardiovascular FunctionOxidative StressPkc-β-dependent PhosphorylationMetabolic Syndrome	2009	81
Voltage-dependent K<sup>+</sup> channels regulate the duration of reactive hyperemia in the canine coronary circulation Gregory M. Dick, Ian N. Bratz, Léna Borbouse, American Journal of Physiology-Heart and Circulatory Physiology	2008	76
Epidermal growth factor‐containing wound closure enhances wound healing in non‐diabetic and diabetic rats Salih Doğan, Seher Demirer, İlknur Kepenekçi, International Wound Journal	2009	73
Coronary arteriolar vasoconstriction to angiotensin II is augmented in prediabetic metabolic syndrome via activation of AT<sub>1</sub> receptors Cuihua Zhang, Jarrod D. Knudson, Srinath Setty, American Journal of Physiology-Heart and Circulatory Physiology HypertensionVascular DiseaseCardiovascular PharmacologyAng IiCardiovascular Function	2005	67

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