Ian N. Bratz - Concepedia

Author

Ian N. Bratz

Northeast Ohio Medical University ORCID

Also Known As

Bratz, Ian N., I. N. Bratz, IAN N. BRATZ, Ian Bratz, Ian N Bratz, Ian N. Bratz, Ian Nathaniel Bratz

Publications

1.6K

Citations

H-Index

Concepts

All Affiliations

Northeast Ohio Medical University Kent State University Indiana University – Purdue University Indianapolis Louisiana State University Health Sciences Center New Orleans Ohio University

About

Ian N. Bratz is an author at Northeast Ohio Medical University specializing in vascular biology, medicine, and physiology.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	13	13	720
2	Health Sciences	7	7	462
3	Biochemistry	5	5	389
4	Cell Biology	3	3	163
5	Applied Physiology	1	1	47

Ian N. Bratz

Publications

1.5K

Citations

H-Index

Page 1

	Year	Citations
Impaired capsaicin-induced relaxation of coronary arteries in a porcine model of the metabolic syndrome Ian N. Bratz, Gregory M. Dick, Johnathan D. Tune, American Journal of Physiology-Heart and Circulatory Physiology Coronary ArteriesMetabolic SyndromeCapsaicin-induced RelaxationCardiovascular Translational ResearchCardiovascular Disease	2008	135
H<sub>2</sub>O<sub>2</sub>-induced redox-sensitive coronary vasodilation is mediated by 4-aminopyridine-sensitive K<sup>+</sup>channels Paul A. Rogers, Gregory M. Dick, Jarrod D. Knudson, American Journal of Physiology-Heart and Circulatory Physiology	2006	107
PKCα mediates acetylcholine-induced activation of TRPV4-dependent calcium influx in endothelial cells Ravi K. Adapala, Phani K. Talasila, Ian N. Bratz, American Journal of Physiology-Heart and Circulatory Physiology Endothelial CellsSpecific Trpv4 AntagonistCardiovascular FunctionCellular PhysiologyIntegrative Physiology	2011	99
Exercise training decreases store-operated Ca2+entry associated with metabolic syndrome and coronary atherosclerosis John M. Edwards, Z. P. Neeb, M. Alloosh, Cardiovascular Research	2009	98
Requisite Role of Kv1.5 Channels in Coronary Metabolic Dilation Vahagn Ohanyan, Liya Yin, Raffi Bardakjian, Circulation Research	2015	97
H<sub>2</sub>O<sub>2</sub> activates redox- and 4-aminopyridine-sensitive K<sub>v</sub> channels in coronary vascular smooth muscle Paul A. Rogers, William M. Chilian, Ian N. Bratz, American Journal of Physiology-Heart and Circulatory Physiology Cardiovascular FunctionMm HRedox BiologyCellular PhysiologyOxidative Stress	2006	97
Impaired function of coronary BK<sub>Ca</sub>channels in metabolic syndrome Léna Borbouse, Gregory M. Dick, Shinichi Asano, American Journal of Physiology-Heart and Circulatory Physiology	2009	90
TRPA1 is functionally co-expressed with TRPV1 in cardiac muscle: Co-localization at z-discs, costameres and intercalated discs Spencer R. Andrei, Pritam Sinharoy, Ian N. Bratz, Channels	2016	79
Voltage-dependent K<sup>+</sup> channels regulate the duration of reactive hyperemia in the canine coronary circulation Gregory M. Dick, Ian N. Bratz, Léna Borbouse, American Journal of Physiology-Heart and Circulatory Physiology	2008	76
Disruption of TRPV1-mediated coupling of coronary blood flow to cardiac metabolism in diabetic mice: role of nitric oxide and BK channels G Guarini, Vahagn Ohanyan, John Kmetz, American Journal of Physiology-Heart and Circulatory Physiology Bk ChannelsNitric OxideTrpv1 ChannelsCardiovascular PharmacologyCardiovascular Function	2012	63

Page 1