Brian D. Hitt - Concepedia

Author

Brian D. Hitt

University of California, Irvine ORCID

Also Known As

Brian D. Hitt, Brian D Hitt, Brian D. Hitt, Brian Hitt, Brian d. Hitt, Hitt, Brian D.

Publications

1.7K

Citations

H-Index

Concepts

All Affiliations

University of California, Irvine Northwestern University The University of Texas Southwestern Medical Center Southwestern Medical Center

About

Brian D. Hitt is an author at University of California, Irvine specializing in cell biology, medicine, and cellular biochemistry.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	4	4	623
2	Cell Biology	4	4	623
3	Cellular Physiology	3	3	212
4	Natural Sciences	2	2	442
5	Social Sciences	2	2	181

Brian D. Hitt

Publications

1.6K

Citations

H-Index

Page 1

	Year	Citations
Phosphorylation of the Translation Initiation Factor eIF2α Increases BACE1 Levels and Promotes Amyloidogenesis Tracy O’Connor, Katherine R. Sadleir, Erika Maus, Neuron Signal TransductionProtein ExpressionNatural SciencesMolecular BiologyProtein Misfolding	2008	411
β-Site Amyloid Precursor Protein Cleaving Enzyme 1 Levels Become Elevated in Neurons around Amyloid Plaques: Implications for Alzheimer's Disease Pathogenesis Jie Zhao, Yifan Fu, Marina V. Yasvoina, Journal of Neuroscience	2007	363
SERCA pump activity is physiologically regulated by presenilin and regulates amyloid β production Kim N. Green, Angelo Demuro, Yama Akbari, The Journal of Cell Biology	2008	296
Enhanced caffeine‐induced Ca<sup>2+</sup>release in the 3xTg‐AD mouse model of Alzheimer's disease Ian F. Smith, Brian D. Hitt, Kim N. Green, Journal of Neurochemistry Ca2+ HomeostasisSynaptic TransmissionCellular PhysiologySocial SciencesAlzheimer's Disease	2005	162
β-Site Amyloid Precursor Protein (APP)-cleaving Enzyme 1 (BACE1)-deficient Mice Exhibit a Close Homolog of L1 (CHL1) Loss-of-function Phenotype Involving Axon Guidance Defects Brian D. Hitt, Sean M. Riordan, Lokesh Kukreja, Journal of Biological Chemistry	2012	146
BACE1-/- mice exhibit seizure activity that does not correlate with sodium channel level or axonal localization Brian D. Hitt, Thomas C. Jaramillo, Dane M. Chetkovich, Molecular Neurodegeneration	2010	96
Ultrasensitive tau biosensor cells detect no seeding in Alzheimer’s disease CSF Brian D. Hitt, Jaime Vaquer‐Alicea, V Manon, Acta Neuropathologica Communications	2021	75
Presenilin regulates capacitative calcium entry dependently and independently of γ-secretase activity Yama Akbari, Brian D. Hitt, M. Paul Murphy, Biochemical and Biophysical Research Communications Capacitative Calcium EntryProtein SecretionSignal TransductionMolecular PhysiologyMedicine	2004	31
CSF-Derived CD4 <sup>+</sup> T-Cell Diversity Is Reduced in Patients With Alzheimer Clinical Syndrome Chaitanya Joshi, Karthigayini Sivaprakasam, Scott Christley, Neurology Neuroimmunology & Neuroinflammation	2021	20
SERCA pump activity is physiologically regulated by presenilin and regulates amyloid β production Kim N. Green, Angelo Demuro, Yama Akbari, The Journal of General Physiology CytoskeletonCellular PhysiologySocial SciencesPhysiological ResearchDegenerative Pathology	2008	19

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