Hamza M. Al-tamari - Concepedia

Author

Hamza M. Al-tamari

Max Planck Institute for Heart and Lung Research ORCID

Also Known As

Hamza M Al-Tamari, Hamza M Al-tamari, Hamza M Al‐Tamari, Hamza M. Al-Tamari, Hamza M. Al-tamari

Publications

737

Citations

H-Index

Concepts

All Affiliations

Max Planck Institute for Heart and Lung Research Justus-Liebig-Universität Gießen Universities of Giessen and Marburg Lung Center German Center for Lung Research

About

Hamza M. Al-tamari is an author at Max Planck Institute for Heart and Lung Research specializing in gene expression, vascular biology, and oxidative stress.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	1	1	277
2	Public Health	1	1	277
3	Cell Biology	1	1	277
4	Immunology	1	1	277
5	Cell Signaling	1	1	277

Hamza M. Al-tamari

Publications

735

Citations

H-Index

Page 1

	Year	Citations
Pro-proliferative and inflammatory signaling converge on FoxO1 transcription factor in pulmonary hypertension Rajkumar Savai, Hamza M. Al-tamari, Daniel Sedding, Nature Medicine InflammationTranscriptional RegulationPulmonary Arterial HypertensionPulmonary CirculationLung Inflammation	2014	277
Macrophage and Cancer Cell Cross-talk via CCR2 and CX3CR1 Is a Fundamental Mechanism Driving Lung Cancer Anja Schmall, Hamza M. Al-tamari, Susanne Herold, American Journal of Respiratory and Critical Care Medicine	2014	265
FoxO3 an important player in fibrogenesis and therapeutic target for idiopathic pulmonary fibrosis Hamza M. Al-tamari, Swati Dabral, Anja Schmall, EMBO Molecular Medicine	2017	118
Effects of phosphodiesterase 4 inhibition on bleomycin-induced pulmonary fibrosis in mice Sergey Udalov, Rio Dumitrascu, Soni Savai Pullamsetti, BMC Pulmonary Medicine	2010	47
Restoration of Megalin-Mediated Clearance of Alveolar Protein as a Novel Therapeutic Approach for Acute Lung Injury Christine U. Vohwinkel, Yasmin Buchäckert, Hamza M. Al-tamari, American Journal of Respiratory Cell and Molecular Biology	2017	18
Depletion of Numb and Numblike in Murine Lung Epithelial Cells Ameliorates Bleomycin-Induced Lung Fibrosis by Inhibiting the β-Catenin Signaling Pathway Alessandro Ianni, Michael Hofmann, Poonam Kumari, Frontiers in Cell and Developmental Biology	2021	10

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