Manzour Hernando Hazbón

Author

Rutgers, The State University of New Jersey ORCID

Also Known As

M H Hazbón, M. Hazbon, M.H. Hazbón, Manzour H. Hazbon, Manzour H. Hazbón, Manzour H. Hazbón, Manzour Hernando Hazbón, Manzour Hernando Hazbón, Manzour H Hazbón

Publications

3.4K

Citations

H-Index

Concepts

All Affiliations

Rutgers, The State University of New Jersey Rutgers New Jersey Medical School Albert Einstein College of Medicine Howard Hughes Medical Institute American Type Culture Collection

About

Manzour Hernando Hazbón is an author at Rutgers, The State University of New Jersey specializing in medicine, microbiology, and clinical microbiology.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	12	12	1.8K
2	Health Sciences	6	6	1.1K
3	Natural Sciences	2	2	138
4	Biochemistry	2	2	441
5	Biology	2	2	83

Manzour Hernando Hazbón

Publications

3.3K

Citations

H-Index

Page 1

	Year	Citations
Global Phylogeny of<i>Mycobacterium tuberculosis</i>Based on Single Nucleotide Polymorphism (SNP) Analysis: Insights into Tuberculosis Evolution, Phylogenetic Accuracy of Other DNA Fingerprinting Systems, and Recommendations for a Minimal Standard SNP Set Ingrid Filliol, Alifiya S. Motiwala, Magali Cavatore, Journal of Bacteriology	2005	434
Population Genetics Study of Isoniazid Resistance Mutations and Evolution of Multidrug-Resistant <i>Mycobacterium tuberculosis</i> Manzour Hernando Hazbón, Michael Brimacombe, Miriam Bobadilla-del-Valle, Antimicrobial Agents and Chemotherapy Isoniazid Resistance MutationsGeneticsTuberculosis PreventionAntibiotic ResistanceDrug Resistance	2006	383
Transfer of a point mutation in Mycobacterium tuberculosis inhA resolves the target of isoniazid Catherine Vilchèze, Feng Wang, Masayoshi Arai, Nature Medicine Pulmonary TuberculosisPathogenesisTuberculosis PreventionMolecular BiologyPoint Mutation	2006	321
Altered NADH/NAD <sup>+</sup> Ratio Mediates Coresistance to Isoniazid and Ethionamide in Mycobacteria Catherine Vilchèze, Torin R. Weisbrod, Bing Chen, Antimicrobial Agents and Chemotherapy Intracellular Nadh/nadBacteriologyMolecular BiologyMicrobial PhysiologyNadh Levels	2005	279
Overexpression of <i>inhA</i>, but not <i>kasA</i>, confers resistance to isoniazid and ethionamide in <i>Mycobacterium smegmatis</i>, <i>M. bovis</i> BCG and <i> M. tuberculosis</i> Michelle H. Larsen, Catherine Vilchèze, Laurent Kremer, Molecular Microbiology	2002	200
The <i>Mycobacterium tuberculosis iniA</i> gene is essential for activity of an efflux pump that confers drug tolerance to both isoniazid and ethambutol Roberto Colangeli, Danica Helb, Sudharsan Sridharan, Molecular Microbiology	2005	182
Phylogenomic analysis of the species of the Mycobacterium tuberculosis complex demonstrates that Mycobacterium africanum, Mycobacterium bovis, Mycobacterium caprae, Mycobacterium microti and Mycobacterium pinnipedii are later heterotypic synonyms of Mycobacterium tuberculosis Marco A. Riojas, Katya J. McGough, Cristin J. Rider-Riojas, INTERNATIONAL JOURNAL OF SYSTEMATIC AND EVOLUTIONARY MICROBIOLOGY	2017	171
Mycothiol biosynthesis is essential for ethionamide susceptibility in <i>Mycobacterium tuberculosis</i> Catherine Vilchèze, Yossef Av‐Gay, Rodgoun Attarian, Molecular Microbiology Pulmonary TuberculosisBiosynthesisHealth SciencesBiochemistryAntibiotics	2008	162
Transcriptional Regulation of Multi-Drug Tolerance and Antibiotic-Induced Responses by the Histone-Like Protein Lsr2 in M. tuberculosis Roberto Colangeli, Danica Helb, Catherine Vilchèze, PLoS Pathogens Drug ToleranceImmunologyBacteriologyMolecular BiologyDrug Resistance	2007	145
Transfer of <i>embB</i> Codon 306 Mutations into Clinical <i>Mycobacterium tuberculosis</i> Strains Alters Susceptibility to Ethambutol, Isoniazid, and Rifampin Hassan Safi, Brendan Sayers, Manzour Hernando Hazbón, Antimicrobial Agents and Chemotherapy	2008	129

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