James N. Topper - Concepedia

Author

James N. Topper

Stanford University

Also Known As

J N Topper, J TOPPER, J.N. Topper, JAMES N. TOPPER, James N Topper, James N. Topper, James N. Topper

Publications

7.9K

Citations

H-Index

Concepts

All Affiliations

Stanford University Brigham and Women's Hospital Millennium Engineering and Integration (United States)Harvard University Beth Israel Deaconess Medical Center

About

James N. Topper is an author at Stanford University specializing in medicine, natural sciences, and gene expression.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	16	16	3.9K
2	Natural Sciences	9	9	857
3	Biochemistry	4	4	543
4	Engineering	3	3	1.3K
5	Health Sciences	2	2	164

James N. Topper

Publications

Citations

H-Index

Page 1

	Year	Citations
The MAD-Related Protein Smad7 Associates with the TGFβ Receptor and Functions as an Antagonist of TGFβ Signaling Hidetoshi Hayashi, Shirin Abdollah, Yubin Qiu, Cell Signal TransductionImmunologyTgfβ ReceptorFibroblast Growth FactorSystems Biology	1997	1.3K
Identification of vascular endothelial genes differentially responsive to fluid mechanical stimuli: cyclooxygenase-2, manganese superoxide dismutase, and endothelial cell nitric oxide synthase are selectively up-regulated by steady laminar shear stress. James N. Topper, Jiexing Cai, D Falb, Proceedings of the National Academy of Sciences Turbulent Shear StressVascular DiseaseEngineeringManganese Superoxide DismutaseRedox Biology	1996	838
Biomechanical activation: an emerging paradigm in endothelial adhesion biology. Michael A. Gimbrone, Tobi Nagel, James N. Topper Journal of Clinical Investigation MechanobiologyEngineeringVascular AdaptationCell AdhesionPhysiology	1997	346
Vascular <i>MAD</i> s: Two novel <i>MAD</i> -related genes selectively inducible by flow in human vascular endothelium James N. Topper, Jiexing Cai, Yubin Qiu, Proceedings of the National Academy of Sciences	1997	323
Promoter selection in human mitochondria involves binding of a transcription factor to orientation-independent upstream regulatory elements Robert P. Fisher, James N. Topper, David A. Clayton Cell Transcriptional RegulationMitochondrial BiogenesisMedicineGeneticsGene Regulation	1987	254
Water‐soluble 17 and 23 kDa polypeptides restore oxygen evolution activity by creating a high‐affinity binding site for Ca<sup>2+</sup> on the oxidizing side of Photosystem II Demetrios F. Ghanotakis, James N. Topper, Ǵerald Babcock, FEBS Letters Oxygen Evolution ActivityPhotorespirationWater‐soluble 17PhotobiologyMolecular Biology	1984	250
CREB binding protein is a required coactivator for Smad-dependent, transforming growth factor β transcriptional responses in endothelial cells James N. Topper, Maria R. DiChiara, Jonathan D. Brown, Proceedings of the National Academy of Sciences	1998	180
Transforming Growth Factor-β1 Inhibition of Macrophage Activation Is Mediated via Smad3 Frank Werner, Mukesh K. Jain, Mark W. Feinberg, Journal of Biological Chemistry	2000	173
Transforming Growth Factor-β1 Inhibits Cytokine-mediated Induction of Human Metalloelastase in Macrophages Frank Werner, Mark W. Feinberg, Mukesh K. Jain, Journal of Biological Chemistry	2000	165
MEKK-1, a Component of the Stress (Stress-activated Protein Kinase/c-Jun N-terminal Kinase) Pathway, Can Selectively Activate Smad2-mediated Transcriptional Activation in Endothelial Cells Jonathan D. Brown, Maria R. DiChiara, Keith R. Anderson, Journal of Biological Chemistry InflammationMolecular PhysiologySignal TransductionAngiogenesisEndothelial Cells	1999	165

Page 1