Frank J.A. Dennissen

Author

German Center for Neurodegenerative Diseases ORCID

Also Known As

F. Dennissen, F.J.A. Dennissen, FJA Dennissen, Frank Dennissen, Frank J A Dennissen, Frank J. A. Dennissen, Frank J.A. Dennissen, Frank JA Dennissen, Frank Johannes Dennissen

Publications

717

Citations

H-Index

Concepts

All Affiliations

German Center for Neurodegenerative Diseases Center of Advanced European Studies and Research Max Planck Institute for Metabolism Research Maastricht University Netherlands Institute for Neuroscience

About

Frank J.A. Dennissen is an author at German Center for Neurodegenerative Diseases specializing in medicine, molecular biology, and neurodegenerative diseases.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	5	5	375
2	Engineering	1	1	37
3	Natural Sciences	1	1	146
4	Allergy	1	1	20
5	Biology	1	1	146

Frank J.A. Dennissen

Publications

574

Citations

H-Index

Page 1

	Year	Citations
Axodendritic sorting and pathological missorting of Tau are isoform-specific and determined by axon initial segment architecture Hans Zempel, Frank J.A. Dennissen, Yatender Kumar, Journal of Biological Chemistry BiologyAxodendritic SortingPattern FormationNatural SciencesMolecular Biology	2017	146
The Tau/A152T mutation, a risk factor for frontotemporal‐spectrum disorders, leads to <scp>NR</scp> 2B receptor‐mediated excitotoxicity Jochen Martin Decker, Lars Krüger, Astrid Sydow, EMBO Reports Mendelian DisorderGenetic DisorderTau/a152t MutationPathologyRisk Factor	2016	133
Adenosine A<sub>1</sub>receptor antagonist rolofylline alleviates axonopathy caused by human Tau ΔK280 Frank J.A. Dennissen, Marta Anglada‐Huguet, Astrid Sydow, Proceedings of the National Academy of Sciences	2016	45
Mutant ubiquitin (UBB<sup>+1</sup>) associated with neurodegenerative disorders is hydrolyzed by ubiquitin C-terminal hydrolase L3 (UCH-L3) Frank J.A. Dennissen, Natalia Kholod, Denise Hermes, FEBS Letters	2011	44
Age-dependent neuroinflammation and cognitive decline in a novel Ala152Thr-Tau transgenic mouse model of PSP and AD Astrid Sydow, Katja Hochgräfe, Stefanie Könen, Acta Neuropathologica Communications	2016	40
Mutant ubiquitin decreases amyloid β plaque formation in a transgenic mouse model of Alzheimer’s disease Paula van Tijn, Frank J.A. Dennissen, Romina J. G. Gentier, Neurochemistry International Neurodegenerative DiseasesAlzheimer's DiseaseMutant Ubiquitin DecreasesMolecular BiologyProtein Degradation	2012	39
Age-related changes of neuron numbers in the frontal cortex of a transgenic mouse model of Alzheimer’s disease Marijke A.M. Lemmens, Annerieke Sierksma, Bart P. F. Rutten, Brain Structure and Function	2011	37
Tau/MAPT disease-associated variant A152T alters tau function and toxicity via impaired retrograde axonal transport Victoria Butler, Dominique A Salazar, David Soriano‐Castell, Human Molecular Genetics EngineeringImmunologyCell DeathPathologyMolecular Biology	2018	37
Long-term proteasomal inhibition in transgenic mice by UBB+1 expression results in dysfunction of central respiration control reminiscent of brainstem neuropathology in Alzheimer patients Martin Irmler, Romina J. G. Gentier, Frank J.A. Dennissen, Acta Neuropathologica	2012	33
Time course of Tau toxicity and pharmacologic prevention in a cell model of Tauopathy Marcus Pickhardt, Jacek Biernat, Sabrina Hübschmann, Neurobiology of Aging AllergyAutoimmune DiseaseCell ModelMedicineTau Toxicity	2017	20

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