Vanida A. Serna - Concepedia

Concepedia

Author

Vanida A. Serna

Northwestern University

Also Known As

V A Serna, V. Serna, V.A. Serna, Vanida A Serna, Vanida A. Serna, Vanida Ann Serna, Vanida Serna

53

Publications

2.2K

Citations

19

H-Index

55

Concepts

All Affiliations

Northwestern University The Ohio State University University at Buffalo, State University of New York Translational Research Institute for Metabolism and Diabetes The Ohio State University Comprehensive Cancer Center – Arthur G. James Cancer Hospital and Richard J. Solove Research Institute

VA

About

Vanida A. Serna is an author at Northwestern University specializing in medicine, cell biology, and cell signaling.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	9	9	1K
2	Cell Biology	8	8	982
3	Public Health	2	2	122
4	Social Sciences	2	2	329
5	Health Sciences	1	1	282

Vanida A. Serna

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23

Publications

2.2K

Citations

19

H-Index

	Year	Citations
Progesterone Is Essential for Maintenance and Growth of Uterine Leiomyoma Hiroshi Ishikawa, K Ishi, Vanida A. Serna, Endocrinology	2010	354
Δ<i>Np63</i> knockout mice reveal its indispensable role as a master regulator of epithelial development and differentiation Rose‐Anne Romano, Kirsten Smalley, Caitlin B.L. Magraw, Development DermatologyCellular PhysiologyTranscriptional RegulationTissue DevelopmentCell Regulation	2012	282
Impaired dopaminergic neurotransmission and microtubule-associated protein tau alterations in human LRRK2 transgenic mice Heather L. Melrose, Justus C. Dächsel, Bahareh Behrouz, Neurobiology of Disease Knockout MouseNeurodegenerative DiseasesSignal TransductionMolecular NeuroscienceMolecular Physiology	2010	276
Paracrine activation of WNT/β-catenin pathway in uterine leiomyoma stem cells promotes tumor growth Masanori Ono, Ping Yin, Antònia Navarro, Proceedings of the National Academy of Sciences	2013	174
Role of Stem Cells in Human Uterine Leiomyoma Growth Masanori Ono, Wenan Qiang, Vanida A. Serna, PLoS ONE	2012	141
Rescue of platinum-damaged oocytes from programmed cell death through inactivation of the p53 family signaling network S-Y Kim, Marília H. Cordeiro, Vanida A. Serna, Cell Death and Differentiation Genome InstabilityCell RegulationApoptosisCell DeathCellular Senescence	2013	127
High-dose saccharin supplementation does not induce gut microbiota changes or glucose intolerance in healthy humans and mice Joan Serrano, Kathleen R. Smith, Audra L. Crouch, Microbiome	2021	104
Transient inhibition of p53 homologs protects ovarian function from two distinct apoptotic pathways triggered by anticancer therapies So‐Youn Kim, Devi M. Nair, Megan M. Romero, Cell Death and Differentiation MedicineApoptosisGynecologyCell DeathAnticancer Therapies	2018	90
Human Uterine Leiomyoma Stem/Progenitor Cells Expressing CD34 and CD49b Initiate Tumors In Vivo Ping Yin, Masanori Ono, Molly B. Moravek, The Journal of Clinical Endocrinology & Metabolism	2015	88
Down-Regulation of miR-29b Is Essential for Pathogenesis of Uterine Leiomyoma Wenan Qiang, Zhaojian Liu, Vanida A. Serna, Endocrinology	2014	71

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