Stefan F. Lichtenthaler

Author

German Center for Neurodegenerative Diseases ORCID

Also Known As

Anna Vlahiotis, Annakaisa Haapasalo, Julie Foley, Lichtenthaler, S, Lichtenthaler, Stefan, Lichtenthaler, Stefan F, Lichtenthaler, Stefan F., Nikolaus Plesnila, Piero Parchi, S Lichtenthaler

323

Publications

12.9K

Citations

H-Index

141

Concepts

All Affiliations

German Center for Neurodegenerative Diseases Munich Cluster for Systems Neurology Technical University of Munich Klinikum rechts der Isar Ludwig-Maximilians-Universität München

About

Stefan F. Lichtenthaler is an author at German Center for Neurodegenerative Diseases specializing in medicine, cell biology, and natural sciences.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	33	57	4.4K
2	Natural Sciences	25	32	2.2K
3	Social Sciences	15	16	1.5K
4	Health Sciences	2	2	136
5	Engineering	1	1	102

Stefan F. Lichtenthaler

137

Publications

9.9K

Citations

H-Index

Page 1

	Year	Citations
ADAM10 is the physiologically relevant, constitutive α‐secretase of the amyloid precursor protein in primary neurons Peer‐Hendrik Kuhn, Huanhuan Wang, Bastian Dislich, The EMBO Journal	2010	592
Phosphorylation of the Translation Initiation Factor eIF2α Increases BACE1 Levels and Promotes Amyloidogenesis Tracy O’Connor, Katherine R. Sadleir, Erika Maus, Neuron Signal TransductionProtein ExpressionNatural SciencesMolecular BiologyProtein Misfolding	2008	411
γ-secretase directly sheds the survival receptor BCMA from plasma cells Sarah Laurent, Franziska Hoffmann, Peer‐Hendrik Kuhn, Nature Communications	2015	353
Cell-type-specific profiling of brain mitochondria reveals functional and molecular diversity Caroline Fecher, Laura Trovò, Stephan A. Müller, Nature Neuroscience Neurodegenerative DiseasesMolecular NeuroscienceMitochondrial FunctionCell-type-specific ProfilingNeurochemical Biomarkers	2019	318
Secretome protein enrichment identifies physiological BACE1 protease substrates in neurons Peer‐Hendrik Kuhn, Katarzyna Koroniak–Szejn, Sebastian Hogl, The EMBO Journal Protein SecretionProtein FunctionSignal TransductionMolecular NeuroscienceMolecular Physiology	2012	312
An Alzheimer‐associated TREM2 variant occurs at the <scp>ADAM</scp> cleavage site and affects shedding and phagocytic function Kai Schlepckow, Gernot Kleinberger, Akio Fukumori, EMBO Molecular Medicine	2017	281
The Cell Adhesion Protein P-selectin Glycoprotein Ligand-1 Is a Substrate for the Aspartyl Protease BACE1 Stefan F. Lichtenthaler, Diana-Ines Dominguez, Gil G. Westmeyer, Journal of Biological Chemistry Protein AssemblyCell AdhesionAspartyl Protease Bace1GlycobiologyImmunology	2003	255
Mechanism of the cleavage specificity of Alzheimer’s disease γ-secretase identified by phenylalanine-scanning mutagenesis of the transmembrane domain of the amyloid precursor protein Stefan F. Lichtenthaler, Rong Wang, Heike S. Grimm, Proceedings of the National Academy of Sciences	1999	231
The E3 Ligase Parkin Maintains Mitochondrial Integrity by Increasing Linear Ubiquitination of NEMO Anne Kathrin Müller-Rischart, Anna Pilsl, Patrick Beaudette, Molecular Cell MitophagyLinear UbiquitinationMitochondrial FunctionNatural SciencesMolecular Biology	2013	212
SPPL2a and SPPL2b promote intramembrane proteolysis of TNFα in activated dendritic cells to trigger IL-12 production Elena Friedmann, Ehud Hauben, Kerstin Maylandt, Nature Cell Biology InflammationCytokineSignal TransductionActivated Dendritic CellsIntramembrane Proteolysis	2006	196

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