Parisha P. Shah - Concepedia

Author

Parisha P. Shah

University of Pennsylvania ORCID

Also Known As

P. P. Shah, P. Shah, Parisha P Shah, Parisha P. Shah, Parisha Shah, Parisha P. Shah, Shah, Parisha P.

Publications

4.5K

Citations

H-Index

Concepts

All Affiliations

University of Pennsylvania University of Glasgow Gladstone Institutes Cancer Research UK Scotland Institute University of California, San Francisco

About

Parisha P. Shah is an author at University of Pennsylvania specializing in medicine, cell biology, and gene expression.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	15	15	2.6K
2	Cell Biology	12	12	2.4K
3	Natural Sciences	5	5	805
4	Engineering	1	1	46
5	Biochemistry	1	1	30

Parisha P. Shah

Publications

3.5K

Citations

H-Index

Page 1

	Year	Citations
Autophagy mediates degradation of nuclear lamina Zhixun Dou, Caiyue Xu, Greg Donahue, Nature Cell AutophagyNuclear LaminaAutophagyProtein DegradationLipophagy	2015	650
Lamin B1 depletion in senescent cells triggers large-scale changes in gene expression and the chromatin landscape Parisha P. Shah, Greg Donahue, Gabriel Otte, Genes & Development Histone ModificationsEpigenetic ChangeEpigeneticsTumor BiologyCell Regulation	2013	544
Lysosome-mediated processing of chromatin in senescence Andre Ivanov, Jeff S. Pawlikowski, Indrani Manoharan, The Journal of Cell Biology	2013	521
Senescent cells harbour features of the cancer epigenome Hazel A. Cruickshanks, Tony McBryan, David M. Nelson, Nature Cell Biology Tumor BiologyCancer GenomicsSenescent CellsMedicineCell Biology	2013	356
Genome-Nuclear Lamina Interactions Regulate Cardiac Stem Cell Lineage Restriction Andrey Poleshko, Parisha P. Shah, Mudit Gupta, Cell ChromatinCardiomyopathyCardiac MuscleDevelopmental BiologyCardiac Reprogramming	2017	222
MLL1 is essential for the senescence-associated secretory phenotype Brian C. Capell, Adam Drake, Jiajun Zhu, Genes & Development Oncogene-induced SenescenceMedicineImmunologyInflammatory ResponseSenescence-associated Secretory Phenotype	2016	143
Pathogenic LMNA variants disrupt cardiac lamina-chromatin interactions and de-repress alternative fate genes Parisha P. Shah, Wenjian Lv, Joshua H. Rhoades, Cell stem cell ChromatinCardiomyopathyCardiac MuscleGeneticsMolecular Genetics	2021	130
Swi2/Snf2-Related Translocases Prevent Accumulation of Toxic Rad51 Complexes during Mitotic Growth Parisha P. Shah, Xiuzhong Zheng, Anastasiya Epshtein, Molecular Cell Toxic Rad51 ComplexesMitotic GrowthCell DivisionNatural SciencesMolecular Biology	2010	110
Biophysical forces mediated by respiration maintain lung alveolar epithelial cell fate Kazushige Shiraishi, Parisha P. Shah, Michael P. Morley, Cell MechanobiologyPulmonary CirculationLung DepositionBiophysical ForcesPhysiology	2023	110
A Balance Between Intermediate Filaments and Microtubules Maintains Nuclear Architecture in the Cardiomyocyte Julie Heffler, Parisha P. Shah, Patrick Robison, Circulation Research	2019	107

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