Yu-Hsi Lin - Concepedia

Author

Yu-Hsi Lin

The University of Texas MD Anderson Cancer Center ORCID

Also Known As

Lin, Yu-Hsi, Y. G Lin, Yu-Hsi Lin

Publications

4.4K

Citations

H-Index

Concepts

All Affiliations

The University of Texas MD Anderson Cancer Center Harvard University Beth Israel Deaconess Medical Center The University of Texas Health Science Center at Houston Henry Ford Hospital

About

Yu-Hsi Lin is an author at The University of Texas MD Anderson Cancer Center specializing in medicine, cell biology, and tumor biology.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	9	9	3.6K
2	Engineering	1	1	48
3	Health Sciences	1	1	15
4	Natural Sciences	1	1	48
5	Biochemistry	1	1	48

Yu-Hsi Lin

Publications

3.9K

Citations

H-Index

Page 1

	Year	Citations
Tumor Evolution of Glioma-Intrinsic Gene Expression Subtypes Associates with Immunological Changes in the Microenvironment Qianghu Wang, Baoli Hu, Xin Hu, Cancer Cell Neuro-oncologyGliomaTumor HeterogeneityImmunologyTumor Immunity	2017	2K
An inhibitor of oxidative phosphorylation exploits cancer vulnerability Jennifer R. Molina, Yuting Sun, Marina Protopopova, Nature Medicine MedicineAnti-cancer AgentCancer VulnerabilityPharmacologyTumor Microenvironment	2018	903
Mutations in the SWI/SNF complex induce a targetable dependence on oxidative phosphorylation in lung cancer Yonathan Lissanu Deribe, Yuting Sun, Christopher Terranova, Nature Medicine Oncogenic AgentMedicineTargetable DependenceOxidative PhosphorylationCancer Genetics	2018	263
KMT2D Deficiency Impairs Super-Enhancers to Confer a Glycolytic Vulnerability in Lung Cancer Hunain Alam, Ming Tang, Mayinuer Maitituoheti, Cancer Cell Oncogenic AgentMedicineCancer Cell BiologyGlycolytic VulnerabilityCancer Genetics	2020	233
SF2312 is a natural phosphonate inhibitor of enolase Paul G. Leonard, Nikunj Satani, David S. Maxwell, Nature Chemical Biology MedicineMechanism Of ActionNatural Phosphonate InhibitorPharmacotherapyChemical Biology	2016	97
An enolase inhibitor for the targeted treatment of ENO1-deleted cancers Yu-Hsi Lin, Nikunj Satani, Naima Hammoudi, Nature Metabolism MedicineEnolase InhibitorPharmacologyPathologyAnti-cancer Agent	2020	95
Enhancer Reprogramming Confers Dependence on Glycolysis and IGF Signaling in KMT2D Mutant Melanoma Mayinuer Maitituoheti, Emily Z. Keung, Ming Tang, Cell Reports	2020	68
Homozygous MTAP deletion in primary human glioblastoma is not associated with elevation of methylthioadenosine Yasaman Barekatain, Jeffrey J. Ackroyd, Victoria C. Yan, Nature Communications	2021	49
Zn(II)-Induced Ground-State π-Deconjugation and Excited-State Electron Transfer in <i>N,N</i>-Bis(2-pyridyl)amino-Substituted Arenes Jye‐Shane Yang, Yan‐Duo Lin, Yu-Hsi Lin, The Journal of Organic Chemistry EngineeringCompound 3DpaExcitation Energy TransferOrganic ChemistryGround-state π-Deconjugation	2004	48
Palladium-Catalyzed Synthesis of <i>trans</i>-4-(<i>N</i>,<i>N</i>-Bis(2-pyridyl)amino)stilbene. A New Intrinsic Fluoroionophore for Transition Metal Ions Jye‐Shane Yang, Yu-Hsi Lin, Ching‐Sheng Yang Organic Letters	2002	47

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