James E. Sligh - Concepedia

Author

James E. Sligh

University of Arizona

Also Known As

J E Sligh, J Sligh, J. Sligh, J.E. Sligh, James E Sligh, James E. Sligh, James E. Sligh, Jr, MD, PhD, James Sligh

Publications

3.3K

Citations

H-Index

Concepts

All Affiliations

University of Arizona Vanderbilt University Medical Center VA Tennessee Valley Healthcare System Southern Arizona VA Health Care System Baylor College of Medicine

About

James E. Sligh is an author at University of Arizona specializing in medicine, cell biology, and dermatology.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	16	21	2.8K
2	Cell Biology	10	11	1.1K
3	Biochemistry	5	5	1.3K
4	Natural Sciences	3	3	1.1K
5	Allergy	2	2	763

James E. Sligh

Publications

3.1K

Citations

H-Index

Page 1

	Year	Citations
The ADP/ATP translocator is not essential for the mitochondrial permeability transition pore Jason E. Kokoszka, Katrina G. Waymire, Shawn Levy, Nature Mitochondrial FunctionBiochemistryNatural SciencesMembrane TransportPhysiology	2004	1.1K
Inflammatory and immune responses are impaired in mice deficient in intercellular adhesion molecule 1. James E. Sligh, Christie M. Ballantyne, Stephen S. Rich, Proceedings of the National Academy of Sciences Cell AdhesionImmunologyImmune RegulationMutant CellsImmune System	1993	562
Overexpression of Akt converts radial growth melanoma to vertical growth melanoma Baskaran Govindarajan, James E. Sligh, Bethaney Vincent, Journal of Clinical Investigation Vertical Growth MelanomaCell RegulationSkin CancerRadial Growth MelanomaRadial Growth	2007	271
Role of the intercellular adhesion molecule-1(ICAM-1) in endotoxin-induced pneumonia evaluated using ICAM-1 antisense oligonucleotides, anti-ICAM-1 monoclonal antibodies, and ICAM-1 mutant mice. Takashi Kumasaka, W M Quinlan, N A Doyle, Journal of Clinical Investigation InflammationIcam-1 MrnaInflammatory Lung DiseaseAllergyLung Inflammation	1996	201
Maternal germ-line transmission of mutant mtDNAs from embryonic stem cell-derived chimeric mice James E. Sligh, Shawn Levy, Katrina G. Waymire, Proceedings of the National Academy of Sciences	2000	125
The roles of CD11/CD18 and ICAM-1 in acute Pseudomonas aeruginosa-induced pneumonia in mice Li Qin, W M Quinlan, N A Doyle, The Journal of Immunology	1996	119
Role of cell defense against oxidative damage in the resistance of Candida albicans to the killing effect of amphotericin B Marcia L. Sokol-Anderson, James E. Sligh, S Elberg, Antimicrobial Agents and Chemotherapy ImmunologyAntimicrobial ChemotherapyOxidative StressDrug ResistanceErgosterol Deficiency	1988	113
Mitochondrial DNA Deletions Serve as Biomarkers of Aging in the Skin, but Are Typically Absent in Nonmelanoma Skin Cancers Alex Eshaghian, Ruth Ann Vleugels, Jeffrey A. Canter, Journal of Investigative Dermatology MedicinePhotocarcinogenesisMelanomaAre Typically AbsentNonmelanoma Skin Cancers	2005	104
Cyclosporine A suppresses keratinocyte cell death through MPTP inhibition in a model for skin cancer in organ transplant recipients Kimberly G. Norman, Jeffrey A. Canter, Mingjian Shi, Mitochondrion OncologyMedicineCutaneous BiologyCell DeathOrgan Transplant Recipients	2009	78
Parkinson's Disease Skin Fibroblasts Display Signature Alterations in Growth, Redox Homeostasis, Mitochondrial Function, and Autophagy Joji Marie Teves, Vedanshi Bhargava, Konner R. Kirwan, Frontiers in Neuroscience MitophagyPathologyRedox HomeostasisDermatologyReactive Oxygen Species	2018	69

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