Hömig-Hölzel Cornelia

Author

Also Known As

Cornelia Hömig-Hölzel, Cornelia Hömig‐Hölzel, Hömig-Hölzel Cornelia, Hömig-Hölzel, Cornelia

Publications

1.1K

Citations

H-Index

Concepts

All Affiliations

Hömig-Hölzel Cornelia is an author at University of Cologne specializing in medicine, immunology, and immunotherapy.

#	Concept	H-Index	Publications	Citations
1	Medicine	3	3	794
2	Immunology	2	2	184
3	Pathology	2	2	184
4	Cell Biology	1	1	13
5	Autoimmune Disease	1	1	13

Hömig-Hölzel Cornelia

Publications

963

Citations

H-Index

Page 1

	Year	Citations
Ultraviolet-radiation-induced inflammation promotes angiotropism and metastasis in melanoma Tobias Bald, Thomas Quast, Jennifer Landsberg, Nature InflammationPhotocarcinogenesisMelanomaDermatologyMedicine	2014	610
Immune Surveillance and Therapy of Lymphomas Driven by Epstein-Barr Virus Protein LMP1 in a Mouse Model Baochun Zhang, Sven Kracker, Tomoharu Yasuda, Cell Lymphoid NeoplasiaImmunologyPathologyImmune SurveillanceAutoimmunity	2012	171
LMP1 signaling can replace CD40 signaling in B cells in vivo and has unique features of inducing class-switch recombination to IgG1 Rastelli Julia, Hömig-Hölzel Cornelia, Jane Seagal, Blood	2007	127
Antagonistic TSC22D1 variants control BRAFE600‐induced senescence Hömig-Hölzel Cornelia, Remco van Doorn, Celia Vogel, The EMBO Journal	2011	27
Near‐genomewide <scp>RNA</scp> i screening for regulators of <scp> BRAF <sup>V</sup> </scp> <sup>600E</sup> ‐induced senescence identifies <i> <scp>RASEF</scp> </i> , a gene epigenetically silenced in melanoma Joanna Kaplon, Hömig-Hölzel Cornelia, Linda Gao, Pigment Cell & Melanoma Research	2014	15
B-cell Expansion and Lymphomagenesis Induced by Chronic CD40 Signaling Is Strictly Dependent on CD19 Caroline Hojer, Samantha Frankenberger, Lothar J. Strobl, Cancer Research Lymphocyte DevelopmentTnf Receptor FamilyImmunologyImmune RegulationPathology	2014	13

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