Ronald G. Gregg - Concepedia

Author

Ronald G. Gregg

University of Louisville ORCID

Also Known As

, Gregg, Ronald G, Gregg, Ronald G., R G Gregg, R Gregg, R. G. Gregg, R. G. S. Gregg, R. Gregg, R.G. GREGG, R.G. Gregg

184

Publications

9.8K

Citations

H-Index

136

Concepts

All Affiliations

University of Louisville University of Wisconsin–Madison Cleveland Eye Clinic Cleveland Clinic Case Western Reserve University

About

Ronald G. Gregg is an author at University of Louisville specializing in medicine, genetics, and molecular physiology.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	31	52	5.1K
2	Natural Sciences	11	12	523
3	Health Sciences	10	10	399
4	Engineering	1	1	15
5	Public Health	1	1	15

Ronald G. Gregg

105

Publications

9.6K

Citations

H-Index

Page 1

	Year	Citations
Insertion of DNA sequences into the human chromosomal β-globin locus by homologous recombination Oliver Smithies, Ronald G. Gregg, Sallie S. Boggs, Nature Chromosome DynamicsGeneticsHomologous RecombinationMolecular BiologyChromosome Biology	1985	990
Targetted correction of a mutant HPRT gene in mouse embryonic stem cells Thomas Doetschman, Ronald G. Gregg, Nobuyo Maeda, Nature Embryonic Stem CellDevelopmental BiologyMutant Hprt GeneMedicineGenetics	1987	790
Targeted deletion in astrocyte intermediate filament (Gfap) alters neuronal physiology. Maureen A. McCall, Ronald G. Gregg, Richard R. Behringer, Proceedings of the National Academy of Sciences Synaptic TransmissionCellular NeurobiologySocial SciencesExperimental NeuropathologyNeurology	1996	351
A calcium channel mutation causing hypokalemic periodic paralysis K Jurkat-Rott, Frank Lehmann‐Horn, Alexis Elbaz, Human Molecular Genetics	1994	314
Mutations in RAB39B Cause X-Linked Intellectual Disability and Early-Onset Parkinson Disease with α-Synuclein Pathology Gabrielle R. Wilson, Joe C.H. Sim, Catriona McLean, The American Journal of Human Genetics Neurodegenerative DiseasesGenetic DisorderGeneticsDegenerative DiseaseDegenerative Pathology	2014	268
Mutations in TRPM1 Are a Common Cause of Complete Congenital Stationary Night Blindness Maria M. van Genderen, Mieke M. C. Bijveld, Y. Claassen, The American Journal of Human Genetics Developmental BiologyOphthalmologyGenetic DisorderGeneticsMedicine	2009	237
Absence of the β subunit ( <i>cchb1</i> ) of the skeletal muscle dihydropyridine receptor alters expression of the α <sub>1</sub> subunit and eliminates excitation-contraction coupling Ronald G. Gregg, Albee Messing, Caroline Strube, Proceedings of the National Academy of Sciences	1996	237
A Transient Receptor Potential-Like Channel Mediates Synaptic Transmission in Rod Bipolar Cells Yin Shen, J. Alexander Heimel, Maarten Kamermans, Journal of Neuroscience	2009	228
The<i>nob2</i>mouse, a null mutation in<i>Cacna1f</i>: Anatomical and functional abnormalities in the outer retina and their consequences on ganglion cell visual responses Bo Chang, John R. Heckenlively, Philippa R. Bayley, Visual Neuroscience	2006	205
Genetic heterogeneity in families with hereditary multiple exostoses. April Cook, Wendy H. Raskind, Susan H. Blanton, PubMed	1993	200

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