Fadil Hannan - Concepedia

Author

Fadil Hannan

Also Known As

F HANNAN, F Hannan, F. Hannan, F. M. Hannan, F.M. Hannan, FM Hannan, Fadil Hannan, Fadil M Hannan, Fadil M. Hannan, Hannan, Fadil M.

188

Publications

4.5K

Citations

H-Index

Concepts

All Affiliations

University of Oxford Churchill Hospital Oxford Centre for Diabetes, Endocrinology and Metabolism University of Liverpool University of Ulster

About

Fadil Hannan is an author at University of Oxford specializing in medicine, pathology, and genetics.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	12	13	887
2	Health Sciences	2	2	52
3	Natural Sciences	2	2	154
4	Biochemistry	2	2	154
5	Biology	1	1	22

Fadil Hannan

Publications

2.4K

Citations

H-Index

Page 1

	Year	Citations
Mutations Affecting G-Protein Subunit α<sub>11</sub>in Hypercalcemia and Hypocalcemia M. Andrew Nesbit, Fadil Hannan, Sarah Howles, New England Journal of Medicine	2013	389
Mutations in AP2S1 cause familial hypocalciuric hypercalcemia type 3 M. Andrew Nesbit, Fadil Hannan, Sarah Howles, Nature Genetics Molecular PhysiologyMendelian DisorderGenetic DisorderGeneticsMolecular Genetics	2012	279
Identification of 70 calcium-sensing receptor mutations in hyper- and hypo-calcaemic patients: evidence for clustering of extracellular domain mutations at calcium-binding sites Fadil Hannan, M. Andrew Nesbit, C. Zhang, Human Molecular Genetics	2012	183
Calcium-sensing receptor (CaSR) mutations and disorders of calcium, electrolyte and water metabolism Fadil Hannan, Rajesh V. Thakker Best Practice & Research Clinical Endocrinology & Metabolism Electrolyte DisorderSignal TransductionMolecular PhysiologyBiochemistrySodium Homeostasis	2013	144
Asymmetric activation of the calcium-sensing receptor homodimer Yang Gao, Michael J. Robertson, Sabrina N. Rahman, Nature Molecular PhysiologySignal TransductionG Protein-coupled ReceptorFunctional SelectivityPhysiology	2021	122
Adaptor protein-2 sigma subunit mutations causing familial hypocalciuric hypercalcaemia type 3 (FHH3) demonstrate genotype–phenotype correlations, codon bias and dominant-negative effects Fadil Hannan, Sarah Howles, Angela Rogers, Human Molecular Genetics	2015	86
Familial isolated primary hyperparathyroidism caused by mutations of the MEN1 gene Fadil Hannan, M. Andrew Nesbit, Paul T. Christie, Nature Clinical Practice Endocrinology &#38 Metabolism Mendelian DisorderGenetic DisorderGeneticsParathyroid HormoneParathyroid Disease	2007	83
Familial Hypocalciuric Hypercalcemia Type 1 and Autosomal-Dominant Hypocalcemia Type 1: Prevalence in a Large Healthcare Population Ridge Dershem, Caroline M. Gorvin, Raghu Metpally, The American Journal of Human Genetics GeneticsGenetic EpidemiologyPathologyCalcium-sensing ReceptorDisease Gene Identification	2020	72
The Calcilytic Agent NPS 2143 Rectifies Hypocalcemia in a Mouse Model With an Activating Calcium-Sensing Receptor (CaSR) Mutation: Relevance to Autosomal Dominant Hypocalcemia Type 1 (ADH1) Fadil Hannan, Gerard Walls, Valerie Babinsky, Endocrinology Nps 2143UrologyMolecular PhysiologySignal TransductionRectifies Hypocalcemia	2015	69
Gα11 mutation in mice causes hypocalcemia rectifiable by calcilytic therapy Caroline M. Gorvin, Fadil Hannan, Sarah Howles, JCI Insight	2017	66

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