Tomasz Skórski - Concepedia

Author

Tomasz Skórski

Also Known As

Skorski, T, T Skorski, T Skórski, T. Skorski, T. Skórski, TOMASZ SKORSKI, TOMASZ SKÓRSKI, Tomasz Skorski, Tomasz Skórski

422

Publications

9.3K

Citations

H-Index

Concepts

All Affiliations

Temple University University of Łódź Thomas Jefferson University Sidney Kimmel Cancer Center University of Pennsylvania

About

Tomasz Skórski is an author at Temple University specializing in medicine, cell biology, and tumor biology.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	30	49	4K
2	Natural Sciences	15	15	792
3	Health Sciences	6	6	688
4	Biochemistry	2	2	109
5	Allergy	1	1	128

Tomasz Skórski

107

Publications

7.9K

Citations

H-Index

Page 1

	Year	Citations
Transformation of hematopoietic cells by BCR/ABL requires activation of a PI-3k/Akt-dependent pathway Tomasz Skórski The EMBO Journal Hematological MalignancySignal TransductionCell RegulationHematopoietic CellsMedicine	1997	575
Selective Inhibition of Leukemia Cell Proliferation by BCR-ABL Antisense Oligodeoxynucleotides Cezary Szczylik, Tomasz Skórski, Nicholas C. Nicolaides, Science Mixed-phenotype Acute LeukemiaImmunologyBcr-abl GeneTumor BiologyMyeloid Neoplasia	1991	347
BCR/ABL Regulates Mammalian RecA Homologs, Resulting in Drug Resistance Artur Słupianek, Christoph Schmütte, Gregory Tombline, Molecular Cell Molecular PharmacologySignal TransductionMolecular RegulationMedicineCell Biology	2001	309
BCR/ABL kinase induces self-mutagenesis via reactive oxygen species to encode imatinib resistance Mateusz Koptyra, Rafal Falinski, Michal O. Nowicki, Blood	2006	294
Signal Transducer and Activator of Transcription (STAT)5 Activation by BCR/ABL Is Dependent on Intact Src Homology (SH)3 and SH2 Domains of BCR/ABL and Is Required for Leukemogenesis Malgorzata Nieborowska-Skorska, Mariusz A. Wasik, Artur Słupianek, The Journal of Experimental Medicine Tyrosine KinaseImmunologyCell DeathBcr/abl Src HomologyMyeloid Neoplasia	1999	272
BCR/ABL oncogenic kinase promotes unfaithful repair of the reactive oxygen species–dependent DNA double-strand breaks Michal O. Nowicki, Rafal Falinski, Mateusz Koptyra, Blood	2004	271
Multilevel Dysregulation of STAT3 Activation in Anaplastic Lymphoma Kinase-Positive T/Null-Cell Lymphoma Qian Zhang, Puthryaveett N. Raghunath, Liquan Xue, The Journal of Immunology	2002	261
Fusion Tyrosine Kinases Induce Drug Resistance by Stimulation of Homology-Dependent Recombination Repair, Prolongation of G<sub>2</sub>/M Phase, and Protection from Apoptosis Artur Słupianek, Grażyna Hoser, Ireneusz Majsterek, Molecular and Cellular Biology	2002	204
Ectopic expression of decorin protein core causes a generalized growth suppression in neoplastic cells of various histogenetic origin and requires endogenous p21, an inhibitor of cyclin-dependent kinases. Manas Kumar Santra, David Mann, Edward W. Mercer, Journal of Clinical Investigation PathologyCell ProliferationCytoskeletonCell CycleCell Growth	1997	197
Rac2-MRC-cIII–generated ROS cause genomic instability in chronic myeloid leukemia stem cells and primitive progenitors Margaret Nieborowska-Skorska, Piotr Kopiński, Regina Ray, Blood	2012	184

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