Matthew Schaller - Concepedia

Author

Matthew Schaller

Also Known As

M A Schaller, M Schaller, M. Schaller, Matthew A Schaller, Matthew A. Schaller, Matthew Schaller, Matthew Schaller (197978), Matthew A. Schaller, Schaller, Matthew

137

Publications

4.9K

Citations

H-Index

112

Concepts

All Affiliations

University of Michigan Michigan Medicine Michigan United University of Florida Okayama University

About

Matthew Schaller is an author at University of Michigan specializing in medicine, immunology, and inflammation.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	25	29	2.2K
2	Allergy	7	7	443
3	Engineering	3	3	200
4	Natural Sciences	3	3	371
5	Biochemistry	1	1	103

Matthew Schaller

Publications

4.6K

Citations

H-Index

Page 1

	Year	Citations
TLR3 is an endogenous sensor of tissue necrosis during acute inflammatory events Karen A. Cavassani, Makoto Ishii, Haitao Wen, The Journal of Experimental Medicine	2008	434
Ly6C <sup>Hi</sup> Blood Monocyte/Macrophage Drive Chronic Inflammation and Impair Wound Healing in Diabetes Mellitus Andrew Kimball, Matthew Schaller, Amrita Joshi, Arteriosclerosis Thrombosis and Vascular Biology	2018	201
Epigenetic Changes in Bone Marrow Progenitor Cells Influence the Inflammatory Phenotype and Alter Wound Healing in Type 2 Diabetes Katherine Gallagher, Amrita Joshi, William F. Carson, Diabetes	2014	199
Genomic analyses reveal recurrent mutations in epigenetic modifiers and the JAK–STAT pathway in Sézary syndrome Mark J. Kiel, Anagh A. Sahasrabuddhe, Delphine Rolland, Nature Communications Reveal Recurrent MutationsSs GenomesGeneticsDisease Gene IdentificationEpigenetics	2015	195
Regulation of T Cell Activation by Notch Ligand, DLL4, Promotes IL-17 Production and Rorc Activation Sumanta Mukherjee, Matthew Schaller, Rupak Neupane, The Journal of Immunology Lymphocyte DevelopmentT-regulatory CellImmune RegulationImmunologyNotch Ligand	2009	187
Deletion of TLR3 Alters the Pulmonary Immune Environment and Mucus Production during Respiratory Syncytial Virus Infection Brian D. Rudd, Joost J. Smit, R. A. Flavell, The Journal of Immunology AsthmaLung InflammationInnate Immune SystemImmunologyPulmonary Immune Environment	2006	183
Cytokine Induced Phenotypic and Epigenetic Signatures Are Key to Establishing Specific Macrophage Phenotypes Nicolai A. Kittan, Ronald M. Allen, Abhay Dhaliwal, PLoS ONE	2013	173
The Histone Methyltransferase Setdb2 Modulates Macrophage Phenotype and Uric Acid Production in Diabetic Wound Repair Andrew Kimball, Frank M. Davis, Aaron D. denDekker, Immunity Epigenetic ChangeDiabetesAutophagyEpigenomicsImmunology	2019	159
TLR9 Differentiates Rapidly from Slowly Progressing Forms of Idiopathic Pulmonary Fibrosis Glenda Trujillo, Alessia Meneghin, Kevin R. Flaherty, Science Translational Medicine EngineeringInflammatory Lung DiseaseTlr9 FunctionsLung InflammationAdvanced Lung Disease	2010	155
TLR9 regulates the mycobacteria-elicited pulmonary granulomatous immune response in mice through DC-derived Notch ligand delta-like 4 Toshihiro Ito, Matthew Schaller, Cory M. Hogaboam, Journal of Clinical Investigation Inflammatory Lung DiseaseLung InflammationInnate Immune SystemImmunologyCd4 T Cell Responses	2008	125

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