Johannes Schlöndorff

Author

Also Known As

J Schlöndorff, J. Schlondorff, J.S. Schlondorff, Johannes S Schlöndorff, Johannes S. Schlondorff, Johannes S. Schlöndorff, Johannes SCHLÖNDORFF, Johannes Schlondorff, Johannes Schlöndorff, Schlondorff, Johannes

Publications

4.4K

Citations

H-Index

Concepts

All Affiliations

Harvard University Beth Israel Deaconess Medical Center Memorial Sloan Kettering Cancer Center Boston Children's Hospital Tri-Institutional PhD Program in Chemical Biology

About

Johannes Schlöndorff is an author at Harvard University specializing in medicine, cell biology, and natural sciences.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	17	18	2.3K
2	Cell Biology	14	14	2K
3	Natural Sciences	10	10	1.1K
4	Biochemistry	3	3	164
5	Biology	1	1	98

Johannes Schlöndorff

Publications

3.6K

Citations

H-Index

Page 1

	Year	Citations
Mutations in the formin gene INF2 cause focal segmental glomerulosclerosis Elizabeth Brown, Johannes Schlöndorff, Daniel Becker, Nature Genetics Glomerular DiseaseGlomerulonephritisMendelian DisorderMedicinePathogenesis	2009	439
Evidence for a Role of a Tumor Necrosis Factor-α (TNF-α)-converting Enzyme-like Protease in Shedding of TRANCE, a TNF Family Member Involved in Osteoclastogenesis and Dendritic Cell Survival Lawrence Lum, Brian Wong, Régis Josien, Journal of Biological Chemistry	1999	407
Intracellular maturation and localization of the tumour necrosis factor α convertase (TACE) Johannes Schlöndorff, J. David Becherer, Carl Blobel Biochemical Journal Tumour Necrosis FactorImmunologyPathologyCell DeathTumor Biology	2000	351
Antagonistic Regulation of Actin Dynamics and Cell Motility by TRPC5 and TRPC6 Channels Dequan Tian, Sarah Melissa P Jacobo, David Billing, Science Signaling Trpc6 ChannelsMolecular BiologyCytoskeletonCellular PhysiologyWhereas Trpc6	2010	274
APOL1 kidney disease risk variants cause cytotoxicity by depleting cellular potassium and inducing stress-activated protein kinases Opeyemi A. Olabisi, Jia-Yue Zhang, Lynn VerPlank, Proceedings of the National Academy of Sciences	2015	226
Small Molecule Targets TMED9 and Promotes Lysosomal Degradation to Reverse Proteinopathy Moran Dvela‐Levitt, Maria Kost‐Alimova, Maheswarareddy Emani, Cell Protein FunctionAutoimmune DiseaseMedicineNatural SciencesT-regulatory Cell	2019	191
TRPC6 mutations associated with focal segmental glomerulosclerosis cause constitutive activation of NFAT-dependent transcription Johannes Schlöndorff, Donato del Camino, Robert Carrasquillo, American Journal of Physiology-Cell Physiology PathologyCellular PhysiologyGlomerulonephritisSignaling PathwayCell Regulation	2009	179
Disease-associated mutant α-actinin-4 reveals a mechanism for regulating its F-actin-binding affinity Astrid Weins, Johannes Schlöndorff, Fumihiko Nakamura, Proceedings of the National Academy of Sciences Molecular RegulationMolecular BiologyCytoskeletonCalponin HomologyCellular Physiology	2007	156
Evidence for a Critical Role of the Tumor Necrosis Factor α Convertase (TACE) in Ectodomain Shedding of the p75 Neurotrophin Receptor (p75NTR) Gisela Weskamp, Johannes Schlöndorff, Lawrence Lum, Journal of Biological Chemistry	2004	149
Six members of the mouse forkhead gene family are developmentally regulated. Klaus H. Kaestner, Khui Hung Lee, Johannes Schlöndorff, Proceedings of the National Academy of Sciences GeneticsGenomic MechanismMolecular GeneticsTranscriptional RegulationGene Structure	1993	143

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