Martin H. Schludi - Concepedia

Author

Martin H. Schludi

German Center for Neurodegenerative Diseases

Also Known As

Martin H Schludi, Martin H. Schludi, Martin Schludi, Schludi, Martin Haribert

Publications

1.7K

Citations

H-Index

Concepts

All Affiliations

German Center for Neurodegenerative Diseases Munich Cluster for Systems Neurology Ludwig-Maximilians-Universität München Helmholtz Zentrum München Technical University of Munich

About

Martin H. Schludi is an author at German Center for Neurodegenerative Diseases specializing in medicine, gene expression, and natural sciences.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	4	4	1.1K
2	Natural Sciences	3	3	773
3	Cell Biology	2	2	417
4	Social Sciences	1	1	316
5	Biochemistry	1	1	101

Martin H. Schludi

Publications

1.6K

Citations

H-Index

Page 1

	Year	Citations
Bidirectional transcripts of the expanded C9orf72 hexanucleotide repeat are translated into aggregating dipeptide repeat proteins Kohji Mori, Thomas Arzberger, Friedrich A. Grässer, Acta Neuropathologica Sequence MotifDipeptide Repeat ProteinsNatural SciencesGeneticsGene Structure	2013	490
C9orf72 FTLD/ALS-associated Gly-Ala dipeptide repeat proteins cause neuronal toxicity and Unc119 sequestration Stephanie May, Daniel Hornburg, Martin H. Schludi, Acta Neuropathologica Neurochemical BiomarkersSocial SciencesGa InclusionsDegenerative PathologyNeurology	2014	316
Distribution of dipeptide repeat proteins in cellular models and C9orf72 mutation cases suggests link to transcriptional silencing Martin H. Schludi Acta Neuropathologica Few InclusionsGeneticsGenomic MechanismMolecular BiologyMolecular Genetics	2015	182
Spinal poly-GA inclusions in a C9orf72 mouse model trigger motor deficits and inflammation without neuron loss Martin H. Schludi, Lore Becker, Lillian Garrett, Acta Neuropathologica	2017	122
Poly‐ <scp>GP</scp> in cerebrospinal fluid links <i>C9orf72</i> ‐associated dipeptide repeat expression to the asymptomatic phase of <scp>ALS</scp> / <scp>FTD</scp> Carina Lehmer, Patrick Oeckl, Jochen H. Weishaupt, EMBO Molecular Medicine	2017	118
Cytoplasmic poly-GA aggregates impair nuclear import of TDP-43 in <i>C9orf72</i> ALS/FTLD Bahram Khosravi, Hannelore Hartmann, Stephanie May, Human Molecular Genetics NeurogenomicsGeneticsMolecular BiologySynaptic SignalingTdp Reporter	2016	101
TDP‐43 loss of function inhibits endosomal trafficking and alters trophic signaling in neurons Benjamin M. Schwenk, Hannelore Hartmann, Alperen Serdaroglu, The EMBO Journal	2016	91
Antibodies inhibit transmission and aggregation of <i>C9orf72</i> poly‐ <scp>GA</scp> dipeptide repeat proteins Qihui Zhou, Carina Lehmer, Meike Michaelsen, EMBO Molecular Medicine	2017	86
Loss of <scp>TMEM</scp> 106B potentiates lysosomal and <scp>FTLD</scp> ‐like pathology in progranulin‐deficient mice Georg Werner, Martin H. Schludi, Johannes Gnörich, EMBO Reports	2020	79

Page 1