Hélène Plun‐Favreau

Author

Hélène Plun‐Favreau

Also Known As

Egor Y. Plotnikov, H Plun-Favreau, H. Plun-Favreau, Helene Plun-Favreau, Helene Plun‐Favreau, Hélène Plun-Favreau, Hélène Plun‐Favreau, Lasse Pihlstrom, Plun-Favreau, Helene, Plun-Favreau, Hélène

Publications

10.9K

Citations

H-Index

Concepts

All Affiliations

University College London National Hospital for Neurology and Neurosurgery Inserm University of Reading National Institutes of Health

About

Hélène Plun‐Favreau is an author at University College London specializing in medicine, cell biology, and mitophagy.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	16	17	3.1K
2	Cell Biology	14	14	2.9K
3	Natural Sciences	4	4	1.1K
4	Biochemistry	4	4	1.1K
5	Social Sciences	1	1	170

Hélène Plun‐Favreau

Publications

4.9K

Citations

H-Index

Page 1

	Year	Citations
Loss of function mutations in the gene encoding Omi/HtrA2 in Parkinson's disease Karsten M. Strauss, L. Miguel Martins, Hélène Plun‐Favreau, Human Molecular Genetics MitophagyMitochondrial DysfunctionGeneticsCell DeathFunction Mutations	2005	563
PINK1 cleavage at position A103 by the mitochondrial protease PARL Emma Deas, Hélène Plun‐Favreau, Sonia Gandhi, Human Molecular Genetics Mitochondrial DynamicNeurodegenerative DiseasesMitochondrial FunctionBiochemistryProtein Folding	2010	478
The mitochondrial protease HtrA2 is regulated by Parkinson's disease-associated kinase PINK1 Hélène Plun‐Favreau, Kristina Klupsch, Nicoleta Moisoi, Nature Cell Biology MitophagyBiochemistryMitochondrial FunctionOrganelle DynamicDisease-associated Kinase Pink1	2007	467
The Parkinson's disease–linked proteins Fbxo7 and Parkin interact to mediate mitophagy Victoria Burchell, David E. Nelson, Álvaro Sánchez-Martínez, Nature Neuroscience Cell AutophagyMitophagyNeurodegenerative DiseasesProtein FunctionParkin Interact	2013	325
CLF associates with CLC to form a functional heteromeric ligand for the CNTF receptor complex Greg Elson, Eric Lelièvre, Catherine Guillet, Nature Neuroscience Signal TransductionFunctional Heteromeric LigandG Protein-coupled ReceptorNatural SciencesReceptor (Biochemistry)	2000	252
Finding genetically-supported drug targets for Parkinson’s disease using Mendelian randomization of the druggable genome Catherine S. Storm, Demis A. Kia, Mona Mohammad Almramhi, Nature Communications	2021	204
Identification of Candidate Parkinson Disease Genes by Integrating Genome-Wide Association Study, Expression, and Epigenetic Data Sets Demis A. Kia, David Zhang, Sebastian Guelfi, JAMA Neurology	2021	195
Lysine 27 Ubiquitination of the Mitochondrial Transport Protein Miro Is Dependent on Serine 65 of the Parkin Ubiquitin Ligase Nicol Birsa, Rosalind Norkett, Tobias Wauer, Journal of Biological Chemistry Mitochondrial FunctionMedicineNatural SciencesParkin Ubiquitin LigaseLysine 27	2014	180
Mitochondrial dysfunction triggered by loss of HtrA2 results in the activation of a brain-specific transcriptional stress response Nicoleta Moisoi, Kristina Klupsch, Valentina Fedele, Cell Death and Differentiation Htra2 ResultsReductive StressMitophagyNeurodegenerative DiseasesMitochondrial Function	2008	170
Enhancing nucleotide metabolism protects against mitochondrial dysfunction and neurodegeneration in a PINK1 model of Parkinson’s disease Roberta Tufi, Sonia Gandhi, Inês Pimenta de Castro, Nature Cell Biology Neurodegenerative DiseasesPink1 ModelBiochemistryMitochondrial FunctionMitochondrial Dysfunction	2014	164

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