Idriss M. Bennani-Baı̈ti

Author

Also Known As

I.M. Bennani-Baiti, IDRISS M. BENNANI-BAITI, Idriss BENNANI-BAITI, Idriss Bennani-Baiti, Idriss M Bennani-Baiti, Idriss M Bennani‐Baiti, Idriss M. Bennani-Baiti, Idriss M. Bennani-Baiti (800264), Idriss M. Bennani-Baïti, Idriss M. Bennani-Baı̈ti

Publications

1.2K

Citations

H-Index

Concepts

All Affiliations

St Anna Children's Hospital St. Anna Children's Cancer Research Institute University of Pennsylvania Howard Hughes Medical Institute Medical University of Vienna

About

Idriss M. Bennani-Baı̈ti is an author at St Anna Children's Hospital specializing in medicine, cell biology, and tumor biology.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	6	6	629
2	Cell Biology	5	5	544
3	Natural Sciences	2	2	99
4	Biochemistry	2	2	99
5	Engineering	1	1	14

Idriss M. Bennani-Baı̈ti

Publications

1.1K

Citations

H-Index

Page 1

	Year	Citations
EZH2 is a mediator of EWS/FLI1 driven tumor growth and metastasis blocking endothelial and neuro-ectodermal differentiation Günther Richter, Stephanie Plehm, Annette Fasan, Proceedings of the National Academy of Sciences Histone ModificationsEpigenetic ChangeEzh2 KnockCancer BiologyEpigenetics	2009	304
Lysine-specific demethylase 1 (LSD1/KDM1A/AOF2/BHC110) is expressed and is an epigenetic drug target in chondrosarcoma, Ewing's sarcoma, osteosarcoma, and rhabdomyosarcoma Idriss M. Bennani-Baı̈ti, Isidro Machado, Antonio Llombart‐Bosch, Human Pathology Tumor BiologyMalignant DiseaseOncogenic AgentMedicineEpigenetic Drug Target	2012	130
EWS-FLI1 Suppresses NOTCH-Activated p53 in Ewing's Sarcoma Jozef Ban, Idriss M. Bennani-Baı̈ti, Max Kauer, Cancer Research	2008	111
Galectin-1 Couples Glycobiology to Inflammation in Osteoarthritis through the Activation of an NF-κB–Regulated Gene Network Stefan Toegel, Daniela Weinmann, Sabine André, The Journal of Immunology	2016	86
Chip interacts with diverse homeodomain proteins and potentiates Bicoid activity <i>in vivo</i> Emi Torigoi, Idriss M. Bennani-Baı̈ti, Christina Rosen, Proceedings of the National Academy of Sciences GeneticsGenomic MechanismMolecular GeneticsChemical BiologyTranscriptional Regulation	2000	85
Hypoxia Modulates EWS-FLI1 Transcriptional Signature and Enhances the Malignant Properties of Ewing's Sarcoma Cells <i>In vitro</i> Dave N.T. Aryee, Stephan Niedan, Maximilian Kauer, Cancer Research Transcriptional RegulationMalignant PropertiesOncologyTumor HypoxiaMalignant Disease	2010	70
DNase I-hypersensitive sites I and II of the human growth hormone locus control region are a major developmental activator of somatotrope gene expression Idriss M. Bennani-Baı̈ti, L. Sylvia, Decheng Song, Proceedings of the National Academy of Sciences	1998	68
Intercohort Gene Expression Co-Analysis Reveals Chemokine Receptors as Prognostic Indicators in Ewing's Sarcoma Idriss M. Bennani-Baı̈ti, Aaron Cooper, Elizabeth R. Lawlor, Clinical Cancer Research	2010	62
Physical Linkage of the Human Growth Hormone Gene Cluster and the CD79b (Igβ/B29) Gene Idriss M. Bennani-Baı̈ti, Nancy E. Cooke, Stephen A. Liebhaber Genomics	1998	39
Physical Linkage of the Human Growth Hormone Gene Cluster and the Skeletal Muscle Sodium Channel α-Subunit Gene (SCN4A) on Chromosome 17 Idriss M. Bennani-Baı̈ti, Beverly K. Jones, STEPHEN A. LIEBHABER, Genomics	1995	35

Page 1