Concepedia

Author

Joy Armistead

University of ManitobaORCID

Also Known As

J. Armistead, Joy Armistead, S. Williams

16

Publications

460

Citations

8

H-Index

16

Concepts

All Affiliations

University of ManitobaChildren's Hospital Research Institute of ManitobaCologne Excellence Cluster on Cellular Stress Responses in Aging Associated DiseasesUniversity of Cologne
JA

About

Joy Armistead is an author at University of Manitoba specializing in medicine, pathology, and cell biology.

Top Concepts

#ConceptH-IndexPublicationsCitations

1

Medicine

3

3

153

2

Cell Biology

2

2

140

3

Molecular Genetics

2

2

137

4

Public Health

1

1

13

5

Molecular Medicine

1

1

16

Joy Armistead

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7

Publications

310

Citations

7

H-Index

YearCitations

Mutation of a Gene Essential for Ribosome Biogenesis, EMG1, Causes Bowen-Conradi Syndrome

Joy Armistead, Sunita Khatkar, Britta Meyer,

The American Journal of Human Genetics

2009

124

Hemicentin-1 is an essential extracellular matrix component of the dermal–epidermal and myotendinous junctions

Daniela Welcker, Cornelia Stein, Natália Martins Feitosa,

Scientific Reports

2021

75

NAMPT-derived NAD+ fuels PARP1 to promote skin inflammation through parthanatos cell death

Francisco J. Martínez-Morcillo, Joaquín Cantón-Sandoval, Francisco J. Martínez‐Navarro,

PLoS Biology

2021

53

Mutation of EMG1 causing Bowen–Conradi syndrome results in reduced cell proliferation rates concomitant with G2/M arrest and 18S rRNA processing delay

Joy Armistead, Richard Hemming, Nehal Patel,

BBA Clinical

2014

18

Growth arrest in the ribosomopathy, Bowen–Conradi syndrome, is due to dramatically reduced cell proliferation and a defect in mitotic progression

Joy Armistead, Nehal Patel, Xiaoli Wu,

Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease

2015

16

The adult polyglucosan body disease mutation GBE1 c.1076A>C occurs at high frequency in persons of Ashkenazi Jewish background

A. Hussain, Joy Armistead, Lara Gushulak,

Biochemical and Biophysical Research Communications

2012

13

Entosis and apical cell extrusion constitute a tumor-suppressive mechanism downstream of Matriptase

Joy Armistead, Julia Hatzold, Anna van Roye,

The Journal of Cell Biology

2019

11

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