Johnathan Cooper‐Knock

Author

Also Known As

Carla Caredda, Clifton L. Dalgard, Cooper-Knock, Johnathan, Giuseppe Borghero, Hemali Phatnani, Highley, J. Cooper‐Knock, Jeffrey D. Rothstein, John Cooper- Knock, John Cooper-Knock

178

Publications

8.7K

Citations

H-Index

Concepts

All Affiliations

University of Sheffield King's College London University College London University Medical Center Utrecht University of Manchester

About

Johnathan Cooper‐Knock is an author at University of Sheffield specializing in medicine, neurology, and neurodegenerative diseases.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	15	16	1.5K
2	Engineering	2	2	174
3	Natural Sciences	2	2	282
4	Public Health	1	1	18
5	Social Sciences	1	1	163

Johnathan Cooper‐Knock

Publications

3.8K

Citations

H-Index

Page 1

	Year	Citations
C9ORF72 GGGGCC Expanded Repeats Produce Splicing Dysregulation which Correlates with Disease Severity in Amyotrophic Lateral Sclerosis Johnathan Cooper‐Knock, Joanna J. Bury, Paul R. Heath, PLoS ONE	2015	498
Clinico-pathological features in amyotrophic lateral sclerosis with expansions in C9ORF72 Johnathan Cooper‐Knock, Christopher Hewitt, J. Robin Highley, Brain Amyotrophic Lateral SclerosisNeurological DisorderIntronic ExpansionGeneticsMedicine	2012	334
Sequestration of multiple RNA recognition motif-containing proteins by C9orf72 repeat expansions Johnathan Cooper‐Knock, Matthew J. Walsh, Adrian Higginbottom, Brain	2014	303
Spatiotemporal Proteomic Analysis of Stress Granule Disassembly Using APEX Reveals Regulation by SUMOylation and Links to ALS Pathogenesis Hagai Marmor-Kollet, Aviad Siany, Nancy Kedersha, Molecular Cell Proteomic TechnologyProtein FunctionAmyotrophic Lateral SclerosisProtein FoldingNatural Sciences	2020	253
Antisense RNA foci in the motor neurons of C9ORF72-ALS patients are associated with TDP-43 proteinopathy Johnathan Cooper‐Knock, Adrian Higginbottom, Matthew J. Stopford, Acta Neuropathologica Molecular BiologyGgggcc Repeat ExpansionsSocial SciencesAntisense Rna FociNeurology	2015	163
TMEM106B is a genetic modifier of frontotemporal lobar degeneration with C9orf72 hexanucleotide repeat expansions Michael D. Gallagher, EunRan Suh, Murray Grossman, Acta Neuropathologica Frontotemporal Lobar DegenerationAllelic VariantGenetic DisorderGeneticsMolecular Biology	2014	146
Loss of nuclear <scp>TDP</scp>‐43 in amyotrophic lateral sclerosis (<scp>ALS</scp>) causes altered expression of splicing machinery and widespread dysregulation of <scp>RNA</scp> splicing in motor neurones J. Robin Highley, Janine Kirby, Joeri A. Jansweijer, Neuropathology and Applied Neurobiology	2014	144
Serum miRNAs miR-206, 143-3p and 374b-5p as potential biomarkers for amyotrophic lateral sclerosis (ALS) Rachel Waller, Marta Milo, Johnathan Cooper‐Knock, Neurobiology of Aging EngineeringNeurochemical BiomarkersPotential BiomarkersMirna SignaturesLong Non-coding Rna	2017	142
SRSF1-dependent nuclear export inhibition of C9ORF72 repeat transcripts prevents neurodegeneration and associated motor deficits Guillaume M. Hautbergue, Lydia M. Castelli, Laura Ferraiuolo, Nature Communications	2017	135
Genome-wide identification of the genetic basis of amyotrophic lateral sclerosis Sai Zhang, Johnathan Cooper‐Knock, Annika K. Weimer, Neuron BioinformaticsGenome-wide Association StudyNeurodegenerative DiseasesAmyotrophic Lateral SclerosisGenetics	2022	116

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