A. van Caam - Concepedia

Author

A. van Caam

Also Known As

A P M van Caam, A van Caam, A. Van Caam, A. van Caam, A.P.M. van Caam, Arjan Caam, Arjan P M van Caam, Arjan P. M. van Caam, Arjan P.M. van Caam, Arjan Petrus Maria van Caam

Publications

1.8K

Citations

H-Index

Concepts

All Affiliations

Radboud University Nijmegen Radboud University Medical Center Radboud Institute for Molecular Life Sciences Leiden University Medical Center Cancer Genomics Centre

About

A. van Caam is an author at Radboud University Nijmegen specializing in medicine, cell biology, and cartilage degeneration.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	9	9	385
2	Cell Biology	8	8	305
3	Immunology	3	3	52
4	Engineering	2	2	106
5	Health Sciences	1	1	80

A. van Caam

Publications

985

Citations

H-Index

Page 1

	Year	Citations
Commercial Cow Milk Contains Physically Stable Extracellular Vesicles Expressing Immunoregulatory TGF-β Bartijn C. H. Pieters, Onno J. Arntz, Miranda B. Bennink, PLoS ONE	2015	208
TGF-β is a potent inducer of Nerve Growth Factor in articular cartilage via the ALK5-Smad2/3 pathway. Potential role in OA related pain? E.N. Blaney Davidson, A. van Caam, E.L. Vitters, Osteoarthritis and Cartilage Musculoskeletal ResearchHealth SciencesPain MedicinePhysiologyAlk5-smad2/3 Pathway	2014	80
Reduced Euchromatin histone methyltransferase 1 causes developmental delay, hypotonia, and cranial abnormalities associated with increased bone gene expression in Kleefstra syndrome mice Monique C.M. Balemans, Muhammad Ansar, Astrid Oudakker, Developmental Biology Histone ModificationsChromatinDevelopmental BiologyChromatin RemodelingGenetic Disorder	2013	75
Physiological and excessive mechanical compression of articular cartilage activates Smad2/3P signaling Wojciech Madej, A. van Caam, E.N. Blaney Davidson, Osteoarthritis and Cartilage InflammationMechanobiologyMusculoskeletal ResearchMolecular PhysiologyEngineering	2014	68
Inducible chondrocyte-specific overexpression of BMP2 in young mice results in severe aggravation of osteophyte formation in experimental OA without altering cartilage damage E.N. Blaney Davidson, E.L. Vitters, M.B. Bennink, Annals of the Rheumatic Diseases	2014	66
TGFβ1-induced SMAD2/3 and SMAD1/5 phosphorylation are both ALK5-kinase-dependent in primary chondrocytes and mediated by TAK1 kinase activity A. van Caam, Wojciech Madej, Amaya García de Vinuesa, Arthritis Research & Therapy	2017	64
Ageing is associated with reduction of mechanically-induced activation of Smad2/3P signaling in articular cartilage Wojciech Madej, A. van Caam, E.N. Blaney Davidson, Osteoarthritis and Cartilage	2015	57
Expression of TGFβ-family signalling components in ageing cartilage: age-related loss of TGFβ and BMP receptors A. van Caam, Wojciech Madej, Eva Thijssen, Osteoarthritis and Cartilage Regenerative MedicineMusculoskeletal ResearchDevelopmental BiologyBone Morphogenic ProteinCartilage Degeneration	2016	41
Unloading results in rapid loss of TGFβ signaling in articular cartilage: role of loading-induced TGFβ signaling in maintenance of articular chondrocyte phenotype? Wojciech Madej, A. van Caam, E. Blaney Davidson, Osteoarthritis and Cartilage Regenerative MedicineMechanobiologyMusculoskeletal ResearchEngineeringBiomechanics	2016	38
The high affinity ALK1-ligand BMP9 induces a hypertrophy-like state in chondrocytes that is antagonized by TGFβ1 A. van Caam, E. Blaney Davidson, Amaya García de Vinuesa, Osteoarthritis and Cartilage Musculoskeletal ResearchMolecular PhysiologyDevelopmental BiologySignal TransductionBone Morphogenic Protein	2015	31

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