Lee Ann Garrett‐Sinha

Author

University at Buffalo, State University of New York ORCID

Also Known As

L A Garrett, L.A. Garrett, LEE ANN GARRETT-SINHA, Lee A. Garrett-Sinha, Lee Ann Garrett, Lee Ann Garrett-Sinha, Lee Ann Garrett‐Sinha, Lee Garrett-Sinha, Lee-Ann Garrett-Sinha, LeeAnn Garrett-Sinha

Publications

3.3K

Citations

H-Index

Concepts

All Affiliations

University at Buffalo, State University of New York Roswell Park Comprehensive Cancer Center University of Chicago Howard Hughes Medical Institute The University of Texas Southwestern Medical Center

About

Lee Ann Garrett‐Sinha is an author at University at Buffalo, State University of New York specializing in medicine, cell biology, and immunology.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	18	19	1.5K
2	Cell Biology	16	17	1.2K
3	Allergy	5	5	331
4	Cellular Physiology	4	4	405
5	Natural Sciences	1	1	37

Lee Ann Garrett‐Sinha

Publications

2.3K

Citations

H-Index

Page 1

	Year	Citations
Inhibition of Oral Mucosal Cell Wound Healing by Bisphosphonates Regina Landesberg, Matthew Cozin, Serge Cremers, Journal of Oral and Maxillofacial Surgery InflammationAnti-inflammatoryMedicineOral CavityWound Care	2008	315
A Gene for a Novel Zinc-finger Protein Expressed in Differentiated Epithelial Cells and Transiently in Certain Mesenchymal Cells Lee Ann Garrett‐Sinha, Heidi Eberspaecher, Michael F. Seldin, Journal of Biological Chemistry Terminal DifferentiationSkin DevelopmentCell LineageDevelopmental BiologyCell Specialization	1996	314
Deletion of ETS-1, a gene in the Jacobsen syndrome critical region, causes ventricular septal defects and abnormal ventricular morphology in mice Maoqing Ye, Chris D. Coldren, Xingqun Liang, Human Molecular Genetics	2009	152
PU.1 and Spi-B Are Required for Normal B Cell Receptor–Mediated Signal Transduction Lee Ann Garrett‐Sinha, Gloria H. Su, Sridhar Rao, Immunity Signal TransductionSpi-b Are RequiredSignaling PathwayAdaptive Immune SystemImmunology	1999	120
The miR-126–VEGFR2 axis controls the innate response to pathogen-associated nucleic acids Judith Agudo, Albert Ruzo, Navpreet Tung, Nature Immunology Transcriptional RegulationPathogen-associated Nucleic AcidsPathogenesisImmunologyRna Biology	2013	119
Ets-1 deficiency leads to altered B cell differentiation, hyperresponsiveness to TLR9 and autoimmune disease Duncheng Wang, Shinu John, James L. Clements, International Immunology Lymphocyte DevelopmentImmunologyImmune RegulationImmunologic MechanismImmunotherapy	2005	118
Environmental sensing by mature B cells is controlled by the transcription factors PU.1 and SpiB Simon N. Willis, Julie Tellier, Yang Liao, Nature Communications	2017	102
IgM and IgD B cell receptors differentially respond to endogenous antigens and control B cell fate Mark Noviski, James L. Mueller, Anne B. Satterthwaite, eLife	2018	95
Ets-1 Regulates Plasma Cell Differentiation by Interfering with the Activity of the Transcription Factor Blimp-1 Shinu John, James L. Clements, Lisa Russell, Journal of Biological Chemistry Immune RegulationImmunologyImmunologic MechanismImmunotherapyInflammation	2007	90
CD28 Promotes Plasma Cell Survival, Sustained Antibody Responses, and BLIMP-1 Upregulation through Its Distal PYAP Proline Motif Henryk Różański, Adam Utley, Louise Carlson, The Journal of Immunology Cd28 ActivationAdaptive Immune SystemImmunologyImmune RegulationCell Death	2015	57

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