Sept4, a Component of Presynaptic Scaffold and Lewy Bodies, Is Required for the Suppression of α-Synuclein Neurotoxicity

Masafumi Ihara, Nobuyuki Yamasaki, Akari Hagiwara, +10

Neuron

Synaptic TransmissionMolecular Biologyα-Synuclein NeurotoxicityCellular NeurobiologySynaptic Signaling +14

The Endoplasmic Reticulum Stress Sensor, ATF6α, Protects against Neurotoxin-induced Dopaminergic Neuronal Death

Naohiro Egawa, Keisuke Yamamoto, Haruhisa Inoue, +4

Journal of Biological Chemistry

ApoptosisCell DeathEr StressSynaptic SignalingRedox Biology +20

Submembranous septins as relatively stable components of actin‐based membrane skeleton

Akari Hagiwara, Yasuhiro Tanaka, Rie Hikawa, +4

Cytoskeleton

CytoskeletonSubmembranous SeptinsOptogeneticsCellular PhysiologySecretory Pathway +15

Differential roles of epigenetic regulators in the survival and differentiation of oligodendrocyte precursor cells

Naohiro Egawa, Akihiro Shindo, Rie Hikawa, +8

Glia

Zonisamide inhibits monoamine oxidase and enhances motor performance and social activity

Maiko Uemura, Takeshi Asano, Rie Hikawa, +2

Neuroscience Research

NeuropsychologySocial ActivityPsychopharmacologyZns-treated MiceSocial Sciences +15

BCAS1-positive immature oligodendrocytes are affected by the α-synuclein-induced pathology of multiple system atrophy

Seiji Kaji, Takakuni Maki, Jun Ueda, +8

Acta Neuropathologica Communications

Homozygous 31 trinucleotide repeats in the SCA2 allele are pathogenic for cerebellar ataxia

Maya Tojima, Gaku Murakami, Rie Hikawa, +4

Neurology Genetics

GeneticsMolecular BiologyPathologyMolecular GeneticsSca2 Allele +15

Cellular analysis of SOD1 protein-aggregation propensity and toxicity: a case of ALS with slow progression harboring homozygous SOD1-D92G mutation

Masanori Sawamura, Keiko Imamura, Rie Hikawa, +13

Scientific Reports

Motor DysfunctionCellular AnalysisProgressive AlsSlow ProgressionExperimental Neuropathology +14