Carlos Bernal‐Mizrachi

Author

Washington University in St. Louis ORCID

Also Known As

C. Bernal-Mizrachi, CARLOS BERNAL-MIZRACHI, Carlos Bernal-Mizrachi, Carlos Bernal-Mizrachi (71315), Carlos Bernal‐Mizrachi

Publications

4.7K

Citations

H-Index

Concepts

All Affiliations

Washington University in St. Louis St. Louis VA Medical Center Saint Louis University KU Leuven

About

Carlos Bernal‐Mizrachi is an author at Washington University in St. Louis specializing in medicine, metabolic syndrome, and physiology.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	13	13	3.1K
2	Health Sciences	8	8	2.1K
3	Biochemistry	4	4	1.5K
4	Public Health	3	3	632
5	Behavioral Sciences	1	1	13

Carlos Bernal‐Mizrachi

Publications

4.4K

Citations

H-Index

Page 1

	Year	Citations
PGC-1α Deficiency Causes Multi-System Energy Metabolic Derangements: Muscle Dysfunction, Abnormal Weight Control and Hepatic Steatosis Teresa C. Leone, John J. Lehman, Brian N. Finck, PLoS Biology Metabolic DisorderPathologyPgc-1alpha NullMitochondrial NumberMetabolic Remodeling	2005	938
Implications of vitamin D deficiency in pregnancy and lactation Megan L. Mulligan, Shaili K. Felton, Amy E. Riek, American Journal of Obstetrics and Gynecology NutritionFertilityLactationClinical NutritionMaternal Health	2009	447
1,25(OH) <sub>2</sub> Vitamin D Inhibits Foam Cell Formation and Suppresses Macrophage Cholesterol Uptake in Patients With Type 2 Diabetes Mellitus Jisu Oh, Sherry Weng, Shaili K. Felton, Circulation	2009	381
Endoplasmic Reticulum Stress Controls M2 Macrophage Differentiation and Foam Cell Formation Jisu Oh, Amy E. Riek, Sherry Weng, Journal of Biological Chemistry InflammationCell AutophagyCell SignalingFoam Cell FormationImmunology	2012	312
A potential link between muscle peroxisome proliferator- activated receptor-α signaling and obesity-related diabetes Brian N. Finck, Carlos Bernal‐Mizrachi, Dong Han, Cell Metabolism ObesityMetabolic SyndromeBiochemistryPhysiologyDiabetes	2005	269
ATM-dependent suppression of stress signaling reduces vascular disease in metabolic syndrome Jochen G. Schneider, Brian N. Finck, Jie Ren, Cell Metabolism Metabolic SyndromeHypertensionVascular DiseaseMetabolic DisorderCardiovascular Disease	2006	237
PPARα deficiency reduces insulin resistance and atherosclerosis in apoE-null mice Karen Tordjman, Carlos Bernal‐Mizrachi, Laura Zemany, Journal of Clinical Investigation Metabolic DisorderImmunologyHyperlipidemiaRobust Atherosclerosis ModelApoe-null Mice	2001	230
Dexamethasone induction of hypertension and diabetes is PPAR-α dependent in LDL receptor–null mice Carlos Bernal‐Mizrachi, Sherry Weng, Feng Chu, Nature Medicine InflammationMolecular PharmacologyHypertensionMetabolic SyndromeHealth Sciences	2003	210
Deletion of Macrophage Vitamin D Receptor Promotes Insulin Resistance and Monocyte Cholesterol Transport to Accelerate Atherosclerosis in Mice Jisu Oh, Amy E. Riek, Isra Darwech, Cell Reports	2015	173
Vascular respiratory uncoupling increases blood pressure and atherosclerosis Carlos Bernal‐Mizrachi, Allison C. Gates, Sherry Weng, Nature HypertensionPulmonary Arterial HypertensionPulmonary CirculationCardiovascular DiseasePhysiology	2005	172

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