Christophe Glorieux - Concepedia

Author

Christophe Glorieux

UCLouvain ORCID

Also Known As

C Glorieux, C. Glorieux, Christophe Glorieux, Christophe Glorieux (146609), GLORIEUX, Christophe, Glorieux, Christophe

Publications

2.9K

Citations

H-Index

Concepts

All Affiliations

UCLouvain Arturo Prat University Sun Yat-sen University Cancer Center Sun Yat-sen University State Key Laboratory of Oncology in South China

About

Christophe Glorieux is an author at UCLouvain specializing in medicine, cell biology, and cancer research.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	14	14	758
2	Cell Biology	10	10	598
3	Biochemistry	3	3	136
4	Natural Sciences	2	2	126
5	Health Sciences	1	1	30

Christophe Glorieux

Publications

1.3K

Citations

H-Index

Page 1

	Year	Citations
Catalase overexpression in mammary cancer cells leads to a less aggressive phenotype and an altered response to chemotherapy Christophe Glorieux, Nicolas Dejeans, Brice Sid, Biochemical Pharmacology Tumor BiologyBreast OncologyOncologyMalignant DiseaseCatalase Overexpression	2011	155
Overexpression of GRP94 in breast cancer cells resistant to oxidative stress promotes high levels of cancer cell proliferation and migration: Implications for tumor recurrence Nicolas Dejeans, Christophe Glorieux, Samuel Guénin, Free Radical Biology and Medicine Cancer Cell ProliferationBreast OncologyMedicineTumor RecurrenceBreast Cancer Cells	2012	96
Regulation of PD-L1 expression in K-ras-driven cancers through ROS-mediated FGFR1 signaling Christophe Glorieux, Xiaojun Xia, Yong-Qiao He, Redox Biology	2020	73
Retinoic acid synergizes ATO-mediated cytotoxicity by precluding Nrf2 activity in AML cells Manuel Valenzuela, Christophe Glorieux, Julie Stockis, British Journal of Cancer	2014	68
Hsp90 Is Cleaved by Reactive Oxygen Species at a Highly Conserved N-Terminal Amino Acid Motif Raphaël Beck, Nicolas Dejeans, Christophe Glorieux, PLoS ONE Hsp90 Protein RadicalsLipid PeroxidationMolecular BiologyReactive Oxygen SpeciesRedox Biology	2012	67
Ascorbate/menadione-induced oxidative stress kills cancer cells that express normal or mutated forms of the oncogenic protein Bcr-Abl. An in vitro and in vivo mechanistic study Raphaël Beck, Rozangela Curi Pedrosa, Nicolas Dejeans, Investigational New Drugs Cancer ResearchOncogenic Protein Bcr-ablOncogenic AgentCell DeathVivo Mechanistic Study	2010	66
Chromatin remodeling regulates catalase expression during cancer cells adaptation to chronic oxidative stress Christophe Glorieux, Juan M. Sandoval, Antoine Fattaccioli, Free Radical Biology and Medicine ChromatinCancer Cells AdaptationRegulates Catalase ExpressionChromatin RemodelingNatural Sciences	2016	59
Catalase down-regulation in cancer cells exposed to arsenic trioxide is involved in their increased sensitivity to a pro-oxidant treatment Christophe Glorieux, Pedro Buc Calderón Cancer Cell International	2018	54
Intracellular ATP levels determine cell death fate of cancer cells exposed to both standard and redox chemotherapeutic agents Julien Verrax, Nicolas Dejeans, Brice Sid, Biochemical Pharmacology MedicineApoptosisCell DeathCancer BiologyCell Death Fate	2011	52
AICAR induces Nrf2 activation by an AMPK-independent mechanism in hepatocarcinoma cells Brice Sid, Christophe Glorieux, Manuel Valenzuela, Biochemical Pharmacology Signal TransductionSignaling PathwayHepatocellular CarcinomaMedicineLiver Physiology	2014	49

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