J Petersen

IL-5-dependent conversion of normodense human eosinophils to the hypodense phenotype uses 3T3 fibroblasts for enhanced viability, accelerated hypodensity, and sustained antibody-dependent cytotoxicity.

M.E. Rothenberg, J Petersen, R L Stevens et al. · The Journal of Immunology · 1989 · 284 citations · Full text

Clinical Immunology, Direct Contact, Immune Regulation +20

Interleukin 5 and phenotypically altered eosinophils in the blood of patients with the idiopathic hypereosinophilic syndrome.

W F Owen, Marc E. Rothenberg, J Petersen et al. · The Journal of Experimental Medicine · 1989 · 227 citations · Full text

Immunodeficiencies, Immunology, Pathology +18

Hypodense eosinophils and interleukin 5 activity in the blood of patients with the eosinophilia-myalgia syndrome.

W F Owen, J Petersen, Daniel M. Sheff et al. · Proceedings of the National Academy of Sciences · 1990 · 95 citations · Full text

Immunology, Extended Viability, Blood Eosinophilia +18

Eosinophils altered phenotypically and primed by culture with granulocyte/macrophage colony-stimulating factor and 3T3 fibroblasts generate leukotriene C4 in response to FMLP.

W F Owen, J Petersen, K. Frank Austen · Journal of Clinical Investigation · 1991 · 35 citations · Full text

Immunology, Cell Culture, Leukotriene C4 +18