David O. Ferguson - Concepedia

Author

David O. Ferguson

Also Known As

D Ferguson, D O Ferguson, D. FERGUSON, D. Ferguson, D. O. Ferguson, D.O. FERGUSON, David Ferguson, David O Ferguson, David O. Ferguson, David Oliver Ferguson

Publications

9.7K

Citations

H-Index

Concepts

All Affiliations

University of Michigan Howard Hughes Medical Institute Harvard University Brigham and Women's Hospital Dana-Farber Cancer Institute

About

David O. Ferguson is an author at University of Michigan specializing in medicine, cell biology, and genome instability.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	29	33	6.1K
2	Natural Sciences	12	12	1.5K
3	Health Sciences	3	3	1.3K
4	Social Sciences	1	1	194
5	Biomolecular Engineering	1	1	197

David O. Ferguson

Publications

8.8K

Citations

H-Index

Page 1

	Year	Citations
Pten dependence distinguishes haematopoietic stem cells from leukaemia-initiating cells Ömer Yılmaz, Riccardo Valdez, Brian Theisen, Nature Developmental BiologyMedicineStem Cell MobilizationHematologyStem Cell Research	2006	1.3K
Interplay of p53 and DNA-repair protein XRCC4 in tumorigenesis, genomic stability and development Yijie Gao, David O. Ferguson, Wei Xie, Nature Tumor BiologyGenome InstabilityChromatinGenomic StabilityMedicine	2000	581
Increased ionizing radiation sensitivity and genomic instability in the absence of histone H2AX Craig H. Bassing, Katrin F. Chua, JoAnn Sekiguchi, Proceedings of the National Academy of Sciences	2002	527
DNA Ligase IV Deficiency in Mice Leads to Defective Neurogenesis and Embryonic Lethality via the p53 Pathway Karen M. Frank, Norman E. Sharpless, Yijie Gao, Molecular Cell Knockout MouseGenome InstabilityDevelopmental BiologyMolecular NeuroscienceMice Leads	2000	497
Histone H2AX Craig H. Bassing, Heikyung Suh, David O. Ferguson, Cell	2003	477
Unrepaired DNA Breaks in p53-Deficient Cells Lead to Oncogenic Gene Amplification Subsequent to Translocations Chengming Zhu, Kevin D. Mills, David O. Ferguson, Cell ChromatinGenome InstabilityP53-deficient Cells LeadOncogenic AgentGenetics	2002	412
The nonhomologous end-joining pathway of DNA repair is required for genomic stability and the suppression of translocations David O. Ferguson, JoAnn Sekiguchi, Sandy Chang, Proceedings of the National Academy of Sciences ChromatinGenome InstabilityDna DamageCell DivisionGenomic Stability	2000	352
Mre11 Nuclease Activity Has Essential Roles in DNA Repair and Genomic Stability Distinct from ATM Activation Jeffrey Buis, Yipin Wu, Yibin Deng, Cell ChromatinGenome InstabilityChromatin RemodelingAtm ActivationNatural Sciences	2008	333
Mouse Model of Colonic Adenoma-Carcinoma Progression Based on Somatic <i>Apc</i> Inactivation Takao Hinoi, Aytekin Akyol, Brian Theisen, Cancer Research	2007	319
ATRX loss promotes tumor growth and impairs nonhomologous end joining DNA repair in glioma Carl Koschmann, Anda‐Alexandra Calinescu, Felipe J. Núñez, Science Translational Medicine	2016	297

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