Tobias Kohl - Concepedia

Author

Tobias Kohl

Ludwig-Maximilians-Universität München ORCID

Also Known As

Kohl, T., Kohl, Tobias, T Kohl, T M Kohl, T. Kohl, Tobias Kohl, Tobias M. Kohl

121

Publications

Citations

H-Index

Concepts

All Affiliations

Ludwig-Maximilians-Universität München Technical University of Munich University of Bonn Max Planck Institute of Experimental Medicine Munich Leukemia Laboratory (Germany)

About

Tobias Kohl is an author at Ludwig-Maximilians-Universität München specializing in medicine, cell biology, and neuroscience.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	11	11	557
2	Health Sciences	3	3	35
3	Social Sciences	3	3	79
4	Engineering	2	2	41
5	Natural Sciences	2	2	88

Tobias Kohl

Publications

1.8K

Citations

H-Index

Page 1

	Year	Citations
KIT-D816 mutations in AML1-ETO-positive AML are associated with impaired event-free and overall survival Susanne Schnittger, Tobias Kohl, Torsten Haferlach, Blood	2005	378
The AML1-ETO fusion gene and the FLT3 length mutation collaborate in inducing acute leukemia in mice Christina Schessl, Vijay P.S. Rawat, Monica Cusan, Journal of Clinical Investigation Hematological MalignancySystems BiologySignal TransductionSignal Transduction PathwaysMedicine	2005	240
A protease assay for two-photon crosscorrelation and FRET analysis based solely on fluorescent proteins Tobias Kohl, Katrin G. Heinze, Rene Kuhlemann, Proceedings of the National Academy of Sciences	2002	134
BH3 mimetic ABT-737 neutralizes resistance to FLT3 inhibitor treatment mediated by FLT3-independent expression of BCL2 in primary AML blasts Tobias Kohl, Christine Hellinger, Farid Ahmed, Leukemia Primary Aml BlastsHematological MalignancyMixed-phenotype Acute LeukemiaFlt3-independent ExpressionImmunology	2007	107
KIT exon 8 mutations associated with core-binding factor (CBF)–acute myeloid leukemia (AML) cause hyperactivation of the receptor in response to stem cell factor Tobias Kohl, Susanne Schnittger, Joachim W. Ellwart, Blood	2004	96
Functional KV10.1 Channels Localize to the Inner Nuclear Membrane Ye Chen‐Izu, Araceli Sánchez, María E. Rubio, PLoS ONE	2011	77
Mutations in the tyrosine kinase domain of FLT3 define a new molecular mechanism of acquired drug resistance to PTK inhibitors in FLT3-ITD–transformed hematopoietic cells Ksenia Bagrintseva, Ruth Schwab, Tobias Kohl, Blood	2003	74
Determining Protease Activity In Vivo by Fluorescence Cross-Correlation Analysis Tobias Kohl, Elke Haustein, Petra Schwille Biophysical Journal Immunocytochemical TechniqueBiochemistryNatural SciencesBioanalysisProtease Activity	2005	72
Ablation of c-FLIP in hepatocytes enhances death-receptor mediated apoptosis and toxic liver injury in vivo Jörn M. Schattenberg, Tim Zimmermann, Marcus A. Wörns, Journal of Hepatology InflammationHepatologyLiver PhysiologyHepatocytes Enhances Death-receptorPathology	2011	63
Diabetic liver injury from streptozotocin is regulated through the caspase-8 homolog cFLIP involving activation of JNK2 and intrahepatic immunocompetent cells Tobias Kohl, Nadine Gehrke, Arno Schad, Cell Death and Disease	2013	47

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