Activation of the Nrf2-regulated antioxidant cell response inhibits HEMA-induced oxidative stress and supports cell viability

Marialucia Gallorini, Christine Petzel, Carola Bolay, +5

Biomaterials

InflammationBiochemistryRedox RegulatorLipid PeroxidationCell Death +7

Resin monomer-induced differential activation of MAP kinases and apoptosis in mouse macrophages and human pulp cells

Stephanie Krifka, Christine Petzel, Karl‐Anton Hiller, +6

Biomaterials

InflammationMap KinasesMedicineApoptosisAutophagy +10

Activation of stress-regulated transcription factors by triethylene glycol dimethacrylate monomer

Stephanie Krifka, Christine Petzel, Carola Bolay, +4

Biomaterials

Transcriptional RegulationMolecular RegulationNatural SciencesStress-regulated Transcription FactorsMolecular Biology +7

2-Hydroxyethyl methacrylate-induced apoptosis through the ATM- and p53-dependent intrinsic mitochondrial pathway

Helmut Schweikl, Christine Petzel, Carola Bolay, +3

Biomaterials

MitophagyMitochondrial FunctionBiochemistryMedicineApoptosis +6

Function of MAPK and downstream transcription factors in monomer-induced apoptosis

Stephanie Krifka, Karl‐Anton Hiller, Carola Bolay, +5

Biomaterials

Signal TransductionSignaling PathwayMolecular PathwayCell RegulationApoptosis +7

Functions of transcription factors NF-κB and Nrf2 in the inhibition of LPS-stimulated cytokine release by the resin monomer HEMA

Helmut Schweikl, Marialucia Gallorini, Gerd Pöschl, +6

Dental Materials

InflammationCytokineImmune Cell DevelopmentLps-stimulated Cytokine ReleaseMedicine +9

Critical role of superoxide anions and hydroxyl radicals in HEMA-induced apoptosis

Helmut Schweikl, M. Godula, Christine Petzel, +3

Dental Materials

InflammationBiochemistryMedicineLipid PeroxidationApoptosis +10

Interaction between LPS and a dental resin monomer on cell viability in mouse macrophages

Helmut Schweikl, Matthias Widbiller, Stephanie Krifka, +4

Dental Materials

InflammationGranulocyteOral CavityImmunologyLymphatic System +8

HEMA-induced oxidative stress inhibits NF-κB nuclear translocation and TNF release from LTA- and LPS-stimulated immunocompetent cells

Helmut Schweikl, Margaritha Birke, Marialucia Gallorini, +5

Dental Materials

InflammationRedox SignalingLps-stimulated Immunocompetent CellsImmune Cell DevelopmentCell Signaling +10

Flavin-containing enzymes as a source of reactive oxygen species in HEMA-induced apoptosis

Helmut Schweikl, Marialucia Gallorini, Markus Forstner, +6

Dental Materials