Carlos Puebla - Concepedia

Author

Carlos Puebla

Pontificia Universidad Católica de Chile ORCID

Also Known As

C Puebla, C. Puebla, Carlos Puebla, Carlos Puebla (316122), Puebla, Carlos

Publications

1.3K

Citations

H-Index

Concepts

All Affiliations

Pontificia Universidad Católica de Chile University of Valparaíso Universitat de Barcelona Centro de Investigación Biomédica en Red University of Chile

About

Carlos Puebla is an author at Pontificia Universidad Católica de Chile specializing in medicine, cellular physiology, and physiology.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	9	9	384
2	Cell Biology	7	7	240
3	Biochemistry	3	3	166
4	Health Sciences	2	2	174
5	Natural Sciences	1	1	22

Carlos Puebla

Publications

841

Citations

H-Index

Page 1

	Year	Citations
De novo expression of connexin hemichannels in denervated fast skeletal muscles leads to atrophy A. Luis, Bruno A. Cisterna, Carlos Puebla, Proceedings of the National Academy of Sciences Muscle FunctionCytoskeletonCellular PhysiologyMuscle AtrophyMolecular Pharmacology	2013	117
Insulin Restores Gestational Diabetes Mellitus–Reduced Adenosine Transport Involving Differential Expression of Insulin Receptor Isoforms in Human Umbilical Vein Endothelium Francisco Westermeier, Carlos Salomón, Marcelo González, Diabetes	2011	110
Active acetylcholine receptors prevent the atrophy of skeletal muscles and favor reinnervation Bruno A. Cisterna, Aníbal A. Vargas, Carlos Puebla, Nature Communications	2020	108
Gestational Diabetes Reduces Adenosine Transport in Human Placental Microvascular Endothelium, an Effect Reversed by Insulin Carlos Salomón, Francisco Westermeier, Carlos Puebla, PLoS ONE	2012	68
Featured Article: Dexamethasone and rosiglitazone are sufficient and necessary for producing functional adipocytes from mesenchymal stem cells David Contador, Fernando Ezquer, Maximiliano Espinosa, Experimental Biology and Medicine Glucocorticoid ReceptorAdipokinesInsulin SignalingRegenerative MedicineMetabolic Syndrome	2015	57
Dexamethasone-induced muscular atrophy is mediated by functional expression of connexin-based hemichannels A. Luis, Elisa Balboa, Carlos Puebla, Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease Molecular PharmacologyMolecular PhysiologySkeletal MuscleFunctional ExpressionMedicine	2016	55
Nitric oxide reduces SLC29A1 promoter activity and adenosine transport involving transcription factor complex hCHOP–C/EBPα in human umbilical vein endothelial cells from gestational diabetes Marcelo Farías, Carlos Puebla, Francisco Westermeier, Cardiovascular Research	2009	53
Nitric oxide reduces adenosine transporter ENT1 gene (<i>SLC29A1</i>) promoter activity in human fetal endothelium from gestational diabetes Marcelo Farías, Rody San Martín, Carlos Puebla, Journal of Cellular Physiology	2006	50
Fast skeletal myofibers of mdx mouse, model of Duchenne muscular dystrophy, express connexin hemichannels that lead to apoptosis A. Luis, Carlos Puebla, Bruno A. Cisterna, Cellular and Molecular Life Sciences Knockout MouseFast Skeletal MyofibersMolecular PhysiologySkeletal MuscleDuchenne Muscular Dystrophy	2016	41
High <scp>D</scp>‐glucose reduces <i>SLC29A1</i> promoter activity and adenosine transport involving specific protein 1 in human umbilical vein endothelium Carlos Puebla, Marcelo Farías, Marcelo González, Journal of Cellular Physiology	2007	30

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