Keith Veitch - Concepedia

Author

Keith Veitch

UCLouvain

Also Known As

K VEITCH, K Veitch, K. VEITCH, K. Veitch, KEITH VEITCH, Keith VEITCH, Keith Veitch

Publications

922

Citations

H-Index

Concepts

All Affiliations

UCLouvain KU Leuven Physiol (Belgium)

About

Keith Veitch is an author at UCLouvain specializing in medicine, physiology, and oxidative stress.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	13	13	565
2	Biochemistry	7	7	395
3	Health Sciences	5	5	130
4	Diabetes	1	1	26
5	Hepatology	1	1	28

Keith Veitch

Publications

759

Citations

H-Index

Page 1

	Year	Citations
Global ischaemia induces a biphasic response of the mitochondrial respiratory chain. Anoxic pre-perfusion protects against ischaemic damage Keith Veitch, ANNICK HOMBROECKX, Danielle Caucheteux, Biochemical Journal Nadh OxidationLipid PeroxidationCell DeathRedox BiologyOxidative Stress	1992	143
In vitro effects of valproate and valproate metabolites on mitochondrial oxidations Sylvie Ponchaut, François Van Hoof, Keith Veitch Biochemical Pharmacology BiochemistryMitochondrial FunctionMitochondrial OxidationsMedicineLipid Peroxidation	1992	105
Role of fructose 2,6-bisphosphate in the control of heart glycolysis C. Depre, Mark H. Rider, Keith Veitch, Journal of Biological Chemistry	1993	79
Flunarizine and cinnarizine inhibit mitochondrial complexes I and II: possible implication for parkinsonism. Keith Veitch, Louis Hue Molecular Pharmacology	1994	49
Cytochrome aa3 depletion is the cause of the deficient mitochondrial respiration induced by chronic valproate administration Sylvie Ponchaut, François Van Hoof, Keith Veitch Biochemical Pharmacology Reductive StressChronic Valproate AdministrationDeficient Mitochondrial RespirationBiochemistryMitochondrial Function	1992	45
Trimetazidine Effects on the Damage to Mitochondrial Functions Caused by Ischemia and Reperfusion Keith Veitch The American Journal of Cardiology Mitochondrial FunctionReperfusion InjuryMedicinePhysiologyMitochondrial Medicine	1995	41
The antianginal agent ranolazine is a weak inhibitor of the respiratory Complex I, but with greater potency in broken or uncoupled than in coupled mitochondria Katrina Wyatt, Caroline Skene, Keith Veitch, Biochemical Pharmacology MedicinePhysiologyMechanism Of ActionAntianginal Agent RanolazinePharmacotherapy	1995	38
Impact of prior or concomitant seasonal influenza vaccination on MF59-adjuvanted H1N1v vaccine (Focetria™) in adult and elderly subjects Roberto Gasparini, F Schioppa, Maria Lattanzi, International Journal of Clinical Practice	2009	37
Altered acyl-CoA metabolism in riboflavin deficiency Keith Veitch, J. P. Draye, Jòseph Vamecq, Biochimica et Biophysica Acta (BBA) - Lipids and Lipid Metabolism Vitamin NutritionBiochemistryPhysiologyRiboflavin DeficiencyCarbonyl Metabolism	1989	34
Comparison of the metabolism of dodecanedioic acid <i>in vivo</i> in control, riboflavin‐deficient and clofibrate‐treated rats Jean‐Pierre Draye, Keith Veitch, Jòseph Vamecq, European Journal of Biochemistry	1988	29

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