Frank A. Fornari - Concepedia

Concepedia

Author

Frank A. Fornari

Virginia Commonwealth University Medical Center

Also Known As

F A Fornari, F. A. Fornari, F.A. Fornari, FRANK A. FORNARI, Frank A. Fornari

18

Publications

1.8K

Citations

14

H-Index

28

Concepts

All Affiliations

Virginia Commonwealth University Medical Center Kettering University Cornell University

FA

About

Frank A. Fornari is an author at Virginia Commonwealth University Medical Center specializing in tumor biology, medicine, and breast cancer.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	7	7	410
2	Health Sciences	2	2	105
3	Engineering	1	1	66
4	Natural Sciences	1	1	66
5	Biochemistry	1	1	66

Frank A. Fornari

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14

Publications

1.8K

Citations

14

H-Index

	Year	Citations
Induction of apoptotic DNA damage and cell death by activation of the sphingomyelin pathway. W. David Jarvis, Richard Kolesnick, Frank A. Fornari, Proceedings of the National Academy of Sciences	1994	455
Interference by doxorubicin with DNA unwinding in MCF-7 breast tumor cells. Frank A. Fornari, Joyce K. Randolph, Jack C. Yalowich, Molecular Pharmacology	1994	440
Attenuation of ceramide-induced apoptosis by diglyceride in human myeloid leukemia cells. W. David Jarvis, Frank A. Fornari, Jeffrey L. Browning, Journal of Biological Chemistry Hematological MalignancyProtein Kinase COncogenic AgentMedicineApoptosis	1994	167
Coordinate Regulation of Stress- and Mitogen-Activated Protein Kinases in the Apoptotic Actions of Ceramide and Sphingosine W. David Jarvis, Frank A. Fornari, Kelly L. Auer, Molecular Pharmacology	1997	152
Induction of Apoptosis and Potentiation of Ceramide-mediated Cytotoxicity by Sphingoid Bases in Human Myeloid Leukemia Cells W. David Jarvis, Frank A. Fornari, Rebecca S. Traylor, Journal of Biological Chemistry	1996	139
Evidence for Involvement of Mitogen-Activated Protein Kinase, Rather than Stress-Activated Protein Kinase, in Potentiation of 1-β-d-Arabinofuranosylcytosine-Induced Apoptosis by Interruption of Protein Kinase C Signaling W. David Jarvis, Frank A. Fornari, Robert M. Tombes, Molecular Pharmacology	1998	89
Induction of differentiation and growth arrest associated with nascent (nonoligosomal) DNA fragmentation and reduced c-myc expression in MCF-7 human breast tumor cells after continuous exposure to a sublethal concentration of doxorubicin. Frank A. Fornari, W. David Jarvis, Steven Grant, PubMed Breast OncologyDna FragmentationCell ProliferationNm DoxorubicinCancer Biology	1994	84
Growth arrest and non-apoptotic cell death associated with the suppression of c-myc Expression in MCF-7 breast tumor cells following acute exposure to doxorubicin Frank A. Fornari, W. David Jarvis, Steven Grant, Biochemical Pharmacology Breast OncologyC-myc ExpressionAcute ExposureApoptosisImmunology	1996	74
Identification and hydropathic characterization of structural features affecting sequence specificity for doxorubicin intercalation into DNA double-stranded polynucleotides Glen E. Kellogg, J.N. Scarsdale, Frank A. Fornari Nucleic Acids Research Bioorganic ChemistryEngineeringDna AnalysisMolecular BiologyIntercalation Site	1998	66
Induction of DNA damage, inhibition of DNA synthesis and suppression of c- myc expression by the anthracycline analog, idarubicin (4-demethoxy-daunorubicin) in the MCF-7 breast tumor cell line David A. Gewirtz, Joyce K. Randolph, Juhi Chawla, Cancer Chemotherapy and Pharmacology Breast OncologyDna DamageOncogenic AgentDna SynthesisBreast Cancer	1998	26

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