Davina J. Hensman Moss

Author

Also Known As

D. Hensman, D. J. Hensman Moss, D.J. Hensman, Davina Hensman, Davina Hensman Moss, Davina Hensman‐Moss, Davina J Hensman Moss, Davina J. Hensman, Davina J. Hensman Moss, Marianne Novak

Publications

2.5K

Citations

H-Index

Concepts

All Affiliations

University College London Cardiff University National Hospital for Neurology and Neurosurgery UK Dementia Research Institute Huntington's Disease Association

About

Davina J. Hensman Moss is an author at University College London specializing in genetics, medicine, and public health.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	3	3	698
2	Public Health	1	1	322
3	Neurology	1	1	343
4	Developmental Biology	1	1	33
5	Epigenetics	1	1	33

Davina J. Hensman Moss

Publications

Citations

H-Index

Page 1

	Year	Citations
Large C9orf72 Hexanucleotide Repeat Expansions Are Seen in Multiple Neurodegenerative Syndromes and Are More Frequent Than Expected in the UK Population Jon Beck, Mark Poulter, Davina J. Hensman Moss, The American Journal of Human Genetics Neurodegenerative DiseasesGenetic DisorderGeneticsMultiple Neurodegenerative SyndromesDegenerative Disease	2013	343
Identification of genetic variants associated with Huntington's disease progression: a genome-wide association study Davina J. Hensman Moss, Antonio F. Pardiñas, Douglas R. Langbehn, The Lancet Neurology Genome-wide Association StudyGenetic AnalysisGenetic VariantsDisease ProgressionGenetics	2017	322
DNA repair pathways underlie a common genetic mechanism modulating onset in polyglutamine diseases Conceição Bettencourt, Davina J. Hensman Moss, Michael Flower, Annals of Neurology	2016	222
<i>C9orf72</i> expansions are the most common genetic cause of Huntington disease phenocopies Davina J. Hensman Moss, Mark Poulter, Jon Beck, Neurology	2013	200
A genetic association study of glutamine-encoding DNA sequence structures, somatic CAG expansion, and DNA repair gene variants, with Huntington disease clinical outcomes Marc Ciosi, Alastair Maxwell, Sarah A. Cumming, EBioMedicine	2019	167
MSH3 modifies somatic instability and disease severity in Huntington’s and myotonic dystrophy type 1 Michael Flower, Vilija Lomeikaite, Marc Ciosi, Brain	2019	161
FAN1 modifies Huntington’s disease progression by stabilizing the expanded <i>HTT</i> CAG repeat Robert Goold, Michael Flower, Davina J. Hensman Moss, Human Molecular Genetics	2018	145
Homozygosity for the C9orf72 GGGGCC repeat expansion in frontotemporal dementia Pietro Fratta, Mark Poulter, Tammaryn Lashley, Acta Neuropathologica	2013	133
Brain Regions Showing White Matter Loss in Huntington’s Disease Are Enriched for Synaptic and Metabolic Genes Peter McColgan, Sarah Gregory, Kiran Seunarine, Biological Psychiatry	2017	102
RNA-Seq of Huntington’s disease patient myeloid cells reveals innate transcriptional dysregulation associated with proinflammatory pathway activation James R. C. Miller, Kitty Lo, Ralph André, Human Molecular Genetics	2016	61

Page 1