Mohammad Reza Ahmadian

Author

Heinrich Heine University Düsseldorf ORCID

Also Known As

Ahmadian, M., Ahmadian, Mohammad R., Ahmadian, Mohammad Reza, M R Ahmadian, M. R. Ahmadian, M. Reza Ahmadian, M.R. Ahmadian, Mohammad Ahmadian, Mohammad R Ahmadian, Mohammad R. Ahmadian

225

Publications

12.4K

Citations

H-Index

150

Concepts

All Affiliations

Heinrich Heine University Düsseldorf Max Planck Institute of Molecular Physiology Max Planck Society Düsseldorf University Hospital Istituto Superiore di Sanità

About

Mohammad Reza Ahmadian is an author at Heinrich Heine University Düsseldorf specializing in medicine, cell biology, and molecular biology.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	37	55	4.1K
2	Natural Sciences	25	33	2.3K
3	Engineering	3	3	189
4	Health Sciences	2	2	123
5	Social Sciences	2	2	77

Mohammad Reza Ahmadian

112

Publications

9.8K

Citations

H-Index

Page 1

	Year	Citations
The Ras-RasGAP Complex: Structural Basis for GTPase Activation and Its Loss in Oncogenic Ras Mutants Klaus Scheffzek, Mohammad Reza Ahmadian, Wolfgang Kabsch, Science	1997	1.5K
CD28-dependent Rac1 activation is the molecular target of azathioprine in primary human CD4+ T lymphocytes Imke Tiede, Gerhard Fritz, Susanne Strand, Journal of Clinical Investigation	2003	801
A restricted spectrum of NRAS mutations causes Noonan syndrome Ion Cristian Cirstea, Kerstin Kutsche, Radovan Dvorský, Nature Genetics Restricted SpectrumMendelian DisorderAutoimmune DiseaseGenetic DisorderMedicine	2009	313
Structural and mechanistic insights into the interaction between Rho and mammalian Dia Rolf Rose, Michael Weyand, Michael Lammers, Nature Protein FunctionBiochemistryProtein AssemblyNatural SciencesBiophysical Aspect	2005	264
The balance of interleukin (IL)-6, IL-6·soluble IL-6 receptor (sIL-6R), and IL-6·sIL-6R·sgp130 complexes allows simultaneous classic and trans-signaling Paul Baran, Selina Hansen, Georg H. Waetzig, Journal of Biological Chemistry	2018	215
Formation of a Transition-State Analog of the Ras GTPase Reaction by Ras·GDP, Tetrafluoroaluminate, and GTPase-Activating Proteins Rohit Mittal, Mohammad Reza Ahmadian, Roger S. Goody, Science Gtpase-activating ProteinsProtein FunctionSignal TransductionBiochemistryRas Gtpase Reaction	1996	202
A novel disorder involving dyshematopoiesis, inflammation, and HLH due to aberrant CDC42 function Michael T. Lam, Simona Coppola, Oliver H.F. Krumbach, The Journal of Experimental Medicine	2019	191
Juvenile myelomonocytic leukemia displays mutations in components of the RAS pathway and the PRC2 network Aurélie Caye, Marion Strullu, Fabien Guidez, Nature Genetics Hematological MalignancyChromatinRas PathwayCell DivisionMedicine	2015	188
Functional Dysregulation of CDC42 Causes Diverse Developmental Phenotypes Simone Martinelli, Oliver H.F. Krumbach, Francesca Pantaleoni, The American Journal of Human Genetics Signal TransductionSignaling PathwayCell RegulationFunctional DysregulationGene Expression	2018	181
Crystal structure of the GTPase-activating domain of human p120GAP and implications for the interaction with Ras Klaus Scheffzek, Alfred Lautwein, Wolfgang Kabsch, Nature Gtpase BiologyCrystal StructureHuman P120gapSignal TransductionMolecular Physiology	1996	175

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