Charles L. Farnsworth

Author

Cell Signaling Technology (United States)

Also Known As

C L Farnsworth, Charles Farnsworth, Charles L. Farnsworth, Charles L. Farnsworth

Publications

Citations

H-Index

Concepts

All Affiliations

Cell Signaling Technology (United States)Tufts University Harvard University Howard Hughes Medical Institute Boston University

About

Charles L. Farnsworth is an author at Cell Signaling Technology (United States) specializing in medicine, cell biology, and natural sciences.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	12	12	1.5K
2	Cell Biology	8	8	1.4K
3	Natural Sciences	7	7	1.1K
4	Biochemistry	7	7	496
5	Health Sciences	2	2	478

Charles L. Farnsworth

Publications

Citations

H-Index

Page 1

	Year	Citations
Calcium activation of Ras mediated by neuronal exchange factor Ras-GRF Charles L. Farnsworth, Norman W. Freshney, Laura Rosen, Nature Molecular PhysiologyNeurophysiologyCalcium ActivationPhysiologyNeurotransmission	1995	426
Molecular cloning of cDNAs encoding a guanine-nucleotide-releasing factor for Ras p21 Chengchao Shou, Charles L. Farnsworth, Benjamin G. Neel, Nature Nucleic Acid ChemistryNatural SciencesMolecular BiologyMolecular CloningGene Expression	1992	361
Regulation of Pluripotency and Cellular Reprogramming by the Ubiquitin-Proteasome System Shannon M. Buckley, Beatriz Aranda-Orgillés, Alexandros Strikoudis, Cell stem cell Protein FunctionCell RegulationCell DivisionMolecular RegulationMedicine	2012	280
Dominant inhibitory mutations in the Mg(2+)-binding site of RasH prevent its activation by GTP. Charles L. Farnsworth, Larry A. Feig Molecular and Cellular Biology Normal Ras FunctionMolecular PharmacologyProtein FunctionSignal TransductionBiochemistry	1991	211
PTMScan Direct: Identification and Quantification of Peptides from Critical Signaling Proteins by Immunoaffinity Enrichment Coupled with LC-MS/MS Matthew P. Stokes, Charles L. Farnsworth, Albrecht Moritz, Molecular & Cellular Proteomics	2012	126
Assessing the Activity of Cediranib, a VEGFR-2/3 Tyrosine Kinase Inhibitor, against VEGFR-1 and Members of the Structurally Related PDGFR Family Sandra R. Brave, Kirsty Ratcliffe, Zena Wilson, Molecular Cancer Therapeutics Vegfr-1 Tyrosine KinaseAngiogenesisMedicineReceptor Tyrosine KinaseCancer Growth	2011	88
Preferential inhibition of the oncogenic form of RasH by mutations in the GAP binding/“effector” domain Charles L. Farnsworth, Mark S. Marshall, Dennis W. Stacey, Cell Oncogenic FormSignal TransductionPreferential InhibitionOncogenic AgentMolecular Biology	1991	82
A mouse homologue of FAST-1 transduces TGFβ superfamily signals and is expressed during early embryogenesis Ellen Weisberg, Glenn E. Winnier, Xin Chen, Mechanisms of Development Mouse HomologueDevelopmental BiologyTissue DevelopmentSignal TransductionMorphogenesis	1998	80
Dominant Inhibitory Mutations in the Mg<sup>2+</sup>-Binding Site of Ras<sup>H</sup> Prevent Its Activation by GTP Charles L. Farnsworth, Larry A. Feig Molecular and Cellular Biology Normal Ras FunctionProtein FunctionSignal TransductionMolecular PhysiologyBiochemistry	1991	70
Widespread protein lysine acetylation in gut microbiome and its alterations in patients with Crohn’s disease Xu Zhang, Zhibin Ning, Janice Mayne, Nature Communications DysbiosisImmunologyGastroenterologyInflammationCrohn ’	2020	68

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