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Ginsenoside Rg3 antagonizes adriamycin-induced cardiotoxicity by improving endothelial dysfunction from oxidative stress via upregulating the Nrf2-ARE pathway through the activation of akt

InflammationAnti-inflammatoryMedicinePhysiologyEndothelial DysfunctionGinsenoside Rg3Vascular BiologyReactive Oxygen SpecieCardiovascular ToxicityPharmacologyCell SignalingOxidative Stress