Publication | Open Access

SMEAF attenuates the production of pro-inflammatory mediators through the inactivation of Akt-dependent NF-κB, p38 and ERK1/2 pathways in LPS-stimulated BV-2 microglial cells

NeuroinflammationInflammationImmune RegulationImmunologyPro-inflammatory MediatorsAkt-dependent Nf-κbErk1/2 PathwaysBrain-immune InteractionInnate ImmunityImmune FunctionMedicineCell BiologyCell SignalingMolecular Signaling