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HO-1 activation contributes to cadmium-induced ferroptosis in renal tubular epithelial cells via increasing the labile iron pool and promoting mitochondrial ROS generation

BiochemistryMedicineMetalloproteinPhysiologyRenal PathologyCadmium-induced FerroptosisBioactive MetalLabile Iron PoolHo-1 Activation ContributesRedox BiologyChronic Kidney DiseaseCell BiologyCell SignalingNephrologyKidney ResearchOxidative Stress