Publication | Open Access

Yellow fever virus infection in human hepatocyte cells triggers an imbalance in redox homeostasis with increased reactive oxygen species production, oxidative stress, and decreased antioxidant enzymes

Viral PersistenceHepatologyViral HepatitisLiver PhysiologyPathogenesisImmunologyViral PathogenesisHepatitisVirologyPathologyHepatotoxicityRedox HomeostasisAntioxidant EnzymesReactive Oxygen SpecieMedicineRedox BiologyOxidative Stress