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Formononetin attenuates high glucose-induced neurotoxicity by negatively regulating oxidative stress and mitochondrial dysfunction in Schwann cells via activation of SIRT3

Mitochondrial DysfunctionMedicineLipid PeroxidationPhysiologySchwann CellsCell DeathMetabolic RegulationNeuroprotectionMetabolismPharmacologyCell BiologyCell SignalingOxidative Stress